An integrative review of personalized feedback interventions for pain and alcohol

Powers, Jessica M.; Zvolensky, Michael J.; Ditre, Joseph W.

doi:10.1016/j.copsyc.2019.01.013

Cited by 7 publications

(2 citation statements)

References 57 publications

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“…Finally, personalized feedback interventions (PFIs) are a subset of motivational interventions that show greater promise for treating co-occurring pain and substance use, and can be delivered via scalable treatment modalities (e.g., computer-delivered interventions, smartphone applications). 161 PFIs motivate behavior change via psychoeducation and presentation of feedback about personal behavior (e.g., risk severity) in normative comparison to others (e.g., from relevant sociodemographic groups). 162 , 163 Parallel lines of inquiry indicate that PFIs decrease maladaptive cognitive-affective and behavioral responses to pain, 164 , 165 and that they reduce hazardous drinking as well as progression to and maintenance of AUD.…”

Section: Discussionmentioning

confidence: 99%

Cognitive-Affective Transdiagnostic Factors Associated With Vulnerability to Alcohol and Prescription Opioid Use in the Context of Pain

Zale

Powers

Ditre

2021

ARCR

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The use of alcohol and prescription opioids is common among people in pain and poses significant public health burdens. This review identifies factors associated with motivation to use alcohol and prescription opioids in the context of pain. Pain-relevant, cognitive-affective, transdiagnostic vulnerability factors—expectancies/motives, pain catastrophizing, pain-related anxiety, distress intolerance, anxiety sensitivity, and perceived interrelations—were selected from theoretical conceptualizations of pain and substance use. Searches conducted in PubMed, PsycINFO, and Embase returned 25 studies that examined associations between identified variables of interest and the use of alcohol and prescription opioids in the context of pain. Consistent with a larger literature on pain and substance use, the studies included in this review demonstrated that people with chronic pain are motivated to use alcohol and opioids in response to negative affect and hold expectancies/motives for coping with pain. Vulnerabilities that engender difficulty managing aversive internal states (distress intolerance and anxiety sensitivity) and maladaptive responses to pain (pain-related anxiety and pain catastrophizing) also were implicated in motivation for alcohol and opioid use. Although one study found that pain-related anxiety was associated with co-use of alcohol and opioids, no studies examined simultaneous use. Future research directions that can explicate causal associations, identify patterns of alcohol and opioid co-use, clarify the role of pain in cessation processes, and inform treatment development are discussed.

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Section: Discussionmentioning

confidence: 99%

Cognitive-Affective Transdiagnostic Factors Associated With Vulnerability to Alcohol and Prescription Opioid Use in the Context of Pain

Zale

Powers

Ditre

2021

ARCR

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“…Despite emerging evidence of comorbidity and reciprocity in terms of a vicious feedback cycle, 6,7 we are not aware of any treatments that have been developed to reduce hazardous drinking or co‐use of alcohol and tobacco in the context of pain. Personalized feedback interventions represent one promising avenue for integrated treatment, in that they are typically brief, easily disseminated, and could be adapted to aid the development of discrepancy between current drinking behavior and desire to mitigate pain 45 …”

Section: Discussionmentioning

confidence: 99%

Pain Severity and Alcohol Use Among Daily Tobacco Cigarette Smokers

2020

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Background and Objectives Pain is associated with hazardous alcohol use. Drinkers have reported using alcohol for pain‐coping, and negative affect may be a key mechanism in pain‐induced motivation to drink. However, no previous study has examined pain severity in relation to alcohol consumption, dependence, and alcohol‐related consequences. Moreover, no studies have examined pain‐alcohol interrelations among tobacco cigarette smokers. These secondary analyses tested the hypotheses that greater past 4‐week pain severity would be positively associated with indices of hazardous drinking (ie, quantity/frequency, harmful use, and dependence), and that the current pain intensity would be positively/indirectly associated with the urge to drink via negative affect. Methods Participants included 225 daily smokers (43% female; MCPD = 22) who completed the baseline session for a larger experimental study. Results Every one‐point increase in pain severity was associated with a 47% increased likelihood of hazardous drinking, and pain severity was positively associated with quantity/frequency of alcohol consumption, harmful patterns of drinking, and alcohol dependence level (Ps < .05). Pain intensity was indirectly associated with urge to drink via negative affect (P < .05). Conclusions These findings provide initial evidence that smokers with greater pain severity may also report hazardous patterns of alcohol use. Scientific Significance This is the first study to demonstrate that past 4‐week pain severity may be one factor that maintains three conceptually distinct patterns of hazardous drinking among smokers. The current results also provide the first evidence that greater pain intensity may be associated with an increased urge to drink alcohol, via negative affect. (Am J Addict 2020;29:134–140)

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Agmatine prevents development of tolerance to anti-nociceptive effect of ethanol in mice

Kotagale¹,

Bhondekar²,

Bhad³

et al. 2022

Alcohol

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An integrative review of personalized feedback interventions for pain and alcohol

Cited by 7 publications

References 57 publications

Cognitive-Affective Transdiagnostic Factors Associated With Vulnerability to Alcohol and Prescription Opioid Use in the Context of Pain

Cognitive-Affective Transdiagnostic Factors Associated With Vulnerability to Alcohol and Prescription Opioid Use in the Context of Pain

Pain Severity and Alcohol Use Among Daily Tobacco Cigarette Smokers

Agmatine prevents development of tolerance to anti-nociceptive effect of ethanol in mice

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