An unusual case of acute disseminated encephalomyelitis confined to brainstem

Fırat, Ahmet; Karakaş, Hakkı Muammer; Yakıncı, Cengiz; Altinok, Tayfun; Alkan, Alpay; Biçak, Uğur

doi:10.1016/j.mri.2004.08.010

Cited by 12 publications

(14 citation statements)

References 7 publications

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“…A common FLAIR finding in both cases was characteristic damage to the bilateral brainstem and the internal capsule. Although the general target areas of ADEM are the corona radiate, periventricular white matter, and basal ganglia, some cases show signal changes in the brainstem and/ or internal capsules [7,8]. Of interest, in the present study the sizes of hyperintense lesions were larger in case 1 than in case 2, particularly in the internal capsule.…”

Section: Discussionmentioning

confidence: 46%

Predicting the motor outcome of acute disseminated encephalomyelitis by apparent diffusion coefficient imaging: Two case reports

Kawashima

Matsukawa

Ueki

et al. 2009

Journal of the Neurological Sciences

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Section: Discussionmentioning

confidence: 46%

Predicting the motor outcome of acute disseminated encephalomyelitis by apparent diffusion coefficient imaging: Two case reports

Kawashima

Matsukawa

Ueki

et al. 2009

Journal of the Neurological Sciences

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“…Firat et al [10] also reported a 6-year-old case with ADEM who had only brainstem involvement. The unusual lesions confined to the mesencephalon responded to steroid therapy [10].…”

Section: Discussionmentioning

confidence: 96%

“…Steroid therapy, not antiviral drugs, was dramatically effective for the mass reduction and symptom improvement [9]. Firat et al [10] also reported a 6-year-old case with ADEM who had only brainstem involvement. The unusual lesions confined to the mesencephalon responded to steroid therapy [10].…”

Section: Discussionmentioning

confidence: 99%

Acute demyelinating encephalomyelitis presenting with a mass lesion

et al. 2015

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Acute disseminated encephalomyelitis (ADEM) is usually a monophasic disease that may follow many infections or vaccines. Neuroimaging plays a key role in the differential diagnosis of ADEM. We reported a female patient with ADEM mimicking a mass lesion in the pons. On physical examination, the patient was drowsy but there were no signs of meningeal irritation. Initial magnetic resonance imaging revealed hyperintense signals on fluid attenuated inversion recovery and T2weighted sequence over distal area of bilateral mesencephalon and pons with involvement of cerebellar pedunculus. The patient was treated intravenous methylprednisolone for 3 days followed by oral prednisolone for another 7 days. The child remained ataxic for 20 days but recovered slowly following 4 months. At the 6th months of follow-up neurological findings of the patient completely recovered, physical examination was normal findings, and control magnetic resonance imaging scanning was also normal. It should be kept in mind that early diagnosis and appropriate treatment of the ADEM could prevent serious neurological sequelae and it is important to make differential diagnosis with other demyelinating diseases and intracranial mass.

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“…64 BS involvement is characterized by single or multiple lesions variable in size which may enhance with intravenous contrast. 65 The MRI abnormalities of ADEM are quite similar to those produced by MS, and these 2 diseases can be indistinguishable. 66,67 Differential diagnosis between these 2 entities is determined on the basis of clinical, MR, and laboratory test results.…”

Section: Disseminating Encephalomyelitismentioning

confidence: 89%

“…69 In MR scans, involvement of the BS together with the cerebellum, basal ganglia, and thalamus is more frequent in ADEM than in MS. 67,68 Although the abnormalities caused by ADEM have sometimes been described as being confined to the BS, more commonly there is concomitant involvement of other areas. 65 Lesions ordinarily affect brain structures bilaterally but asymmetrically and are variable in size, 68 with a tendency for lesions to be large and irregular in shape. 70 Furthermore, in contrast to MS, demyelination zones ordinarily spare the corpus callosum and the periventricular white matter.…”

Section: Disseminating Encephalomyelitismentioning

confidence: 99%

Differential Diagnosis of T2 Hyperintense Brainstem Lesions: Part 1. Focal Lesions

Guzmán-De-Villoria

Fernández-García

Ferreiro-Argüelles

2010

Seminars in Ultrasound, CT and MRI

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Brainstem lesions can be classified as focal or diffuse. Magnetic resonance imaging is the most suitable imaging modality for evaluating these lesions. As a rule, focal lesions are not large and have well-defined margins. Causes include tumors, vascular malformations, demyelinating diseases, brain abscesses, hypertrophic olivary degeneration, and dilated Virchow-Robin spaces. Differential diagnoses of these numerous entities mandates a review of magnetic resonance imaging findings in conjunction with epidemiologic aspects, clinical features, and other medical test results.

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An unusual case of acute disseminated encephalomyelitis confined to brainstem

Cited by 12 publications

References 7 publications

Predicting the motor outcome of acute disseminated encephalomyelitis by apparent diffusion coefficient imaging: Two case reports

Predicting the motor outcome of acute disseminated encephalomyelitis by apparent diffusion coefficient imaging: Two case reports

Acute demyelinating encephalomyelitis presenting with a mass lesion

Differential Diagnosis of T2 Hyperintense Brainstem Lesions: Part 1. Focal Lesions

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