Anencephaly in Uniovular Twins: Report of a Case

Stallybrass, F. C.

doi:10.1136/jech.14.3.136

Cited by 3 publications

(2 citation statements)

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“…Sometimes a different development may be found in uniovular twins, such as hydramnios in one, anencephaly, reticulocytosis, etc., which support the idea th at there are probably additional factors, such as the hemodynamic effects of unequal branching [9] and other environm ental factors which may play a major role in the etiology of different development of uniovular twins [22]. Monoamniotic twin pregnancy is a very rare condition in uniovular twin pregnancy (indeed no such case appears in our records) which may lead to the death of one or both foetus, as a result of mechanical factors [17].…”

Section: Neri Antepartum Death Of One Uniovular Twin 379mentioning

confidence: 56%

Antepartum Death of one Uniovular Twin

Neri¹

1967

Gynecol Obstet Invest

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Section: Neri Antepartum Death Of One Uniovular Twin 379mentioning

confidence: 56%

Antepartum Death of one Uniovular Twin

Neri¹

1967

Gynecol Obstet Invest

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“…This has been interpreted in the past simply as evidence of discordancy in monozygotic twins and as evidence of the importance of non-genetic factors. However, the direct evidence for discordant occurrences in monozygotic pairs depends mainly upon chorionic appearances and they are at best very rare (Stallybrass, 1960) while the fetus-fetus interaction offers a much more specific explanation. Just as singleton anencephalics were supposed to have arisen from twins, so discordant twins were supposed to have derived from initial triplets.…”

Section: Introducrionmentioning

confidence: 99%

Twins and neural tube defects

Knox

1974

Journal of Epidemiology & Community Health

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The hypothesis that anencephalus stems from fetus-fetus interactions in dizygotic twin pairs is examined by comparing the epidemiological predictions of the hypothesis with available observations. The hypothesis itself was based upon the disease-discordance and sex-concordance characteristics of twin pairs affected with anencephalus, and upon the sex ratio of the disease itself. The testable predictions of the hypothesis are (a) that variations in the incidence of anencephalus should be related to variations in dizygotic twinning rates, and particularly that dizygotic twinning rates will set upper limits to the incidence of anencephalus, and (b) that the F/M ratio in anencephalic infants will be high in circumstances of high incidence, and specifically when the incidence is high in relation to the dizygotic twinning rate. Examples from international comparisons, secular changes, social class gradients, and variations according to maternal age confirm a consistent correspondence between observations and these predictions. In addition, the possibility was tested that some fetus-fetus interactions might be based upon sequential rather than simultaneous pairs of fetuses. This model predicted asymmetries of sex ratio in sibs immediately preceding propositi, with differences according to the sex of the affected child, and the predicted findings were confirmed. The fetus-fetus interaction hypothesis is therefore extended in the terms that about one third of occurrences are determined in a sequential manner.

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Mortality and Maldevelopment

Kalter

2007

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Anencephaly in Uniovular Twins: Report of a Case

Cited by 3 publications

References 4 publications

Antepartum Death of one Uniovular Twin

Antepartum Death of one Uniovular Twin

Twins and neural tube defects

Mortality and Maldevelopment

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