Anesthetic effect of CO2 on fish. I. Changes in depth of anesthesia of the carp anesthetized with a constant level of CO2.

Yoshikawa, Hiromasa; Ishida, Yusuke; Ueno, Saburo; Mitsuda, Hisateru

doi:10.2331/suisan.54.457

Cited by 33 publications

(26 citation statements)

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“…Evaluations were made in four consecutive phases (Theinpoint and Niemegeers, 1965;Yoshikawa et al, 1988): (1) Acceleration and subsequent deceleration of opercular movements, a partial loss of reactivity to external stimuli.…”

Section: Acute Toxicity Of 2-phenoxyethanolmentioning

confidence: 99%

Effects of 2-phenoxyethanol anaesthesia on sheatfish (Silurus glanis L.)

Velíšek¹,

Własow²,

Gomułka³

et al. 2007

Vet. Med.

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The aim of the study was to investigate the acute toxicity of 2-phenoxyethanol to sheatfish, and using the values of haematological and biochemical profiles of blood and histological tissue examinations to assess the effects of the fish exposure to that anaesthetic. The values of acute toxicity of 2-phenoxyethanol to sheatfish were found to be 10minLC50 0.77 ml/l, 10minLC0.1 0.42 ml/l, 10minLC99.9 1.90 ml/l, 96hLC50 0.29 ml/l, 96hLC0.1 0.20 ml/l, and 96hLC99.9 0.41 ml/l. The 10-min exposure to 2-phenoxyethanol at a concentration of 0.30 ml/l caused significantly higher values (P < 0.05) of packed cell volume (PCV), mean corpuscular volume (MCV), glucose (GLU) and albumins (ALB) immediately after anaesthesia. A significant decrease (P < 0.05) in the level of alanine aminotransferase (ALT) and a significant increase (P < 0.05) in the values of mean corpuscular haemoglobin concentration (MCHC) were found 24 h post anaesthesia. Histological examinations showed capillary ectasia of gill filaments immediately after 2-phenoxyethanol anaesthesia. Twenty-four hours after anaesthesia, no ectasia was observed. No histopathological changes were demonstrated in other tissues (liver, spleen, cranial and caudal kidneys) following anaesthesia. The results of examinations suggest that the use of 2-phenoxyethanol at a concentration of 0.30 ml/l does not cause any irreversible damage in sheatfish.

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Section: Acute Toxicity Of 2-phenoxyethanolmentioning

confidence: 99%

Effects of 2-phenoxyethanol anaesthesia on sheatfish (Silurus glanis L.)

Velíšek¹,

Własow²,

Gomułka³

et al. 2007

Vet. Med.

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“…Evaluations were made in four consecutive phases (Thienpoint and Niemegeers 1965;Yoshikawa et al 1988):…”

Section: Acute Toxicity Of Clove Oilmentioning

confidence: 99%

Effects of Clove Oil Anaesthesia on European Catfish (Silurus glanis L.)

et al. 2006

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Velí‰ek J., T. Wlasow, P. Gomulka, Z. Svobodová, L. Novotn˘, E. Ziomek: Effects of Clove Oil Anaesthesia on European Catfish (Silurus glanis L.). Acta Vet. Brno 2006, 75: 99-106.The aim of the study was to investigate acute toxicity of clove oil for European catfish and, using values of haematological and biochemical profiles of blood and histological tissue examinations, to assess the effects of the fish exposure to that anaesthetic. Acute toxicity values of clove oil for European catfish found were 10minLC50 76.70 mg·l -1 ; 10minLC0.1 49.60 mg·l -1 ; 10minLC99.9 118.50 mg·l -1 ; 96hLC50 18.40 mg·l -1 ; 96hLC0.1 10.70 mg·l -1 ; and 96hLC99.9 31.90 mg·l -1 . Individual phases of anaesthesia and recovery were determined. The 10-min exposure to clove oil at a concentration of 30 mg·l -1 caused a significant (p < 0.05) increase in the concentration of triacylglycerols (TRIG), alanin aminotransferase (ALT) and decreased (p < 0.05) in mean corpuscular haemoglobin concentration (MCHC) immediately after anaesthesia. The leukocyte counts were significantly (p < 0.05) decreased 24 hours after anaesthesia. A significant (p < 0.05) decrease of percentage distribution lymphocytes was found immediately after anaesthesia. On the other hand, percentage and absolute count of myeloid cells were increased. Increased percentage count of eosinophils outlasted 24 hours after anaesthesia, absolute counts of these cells were consistents with control. Histological examination showed capillary ectasia of gill filaments immediately after clove oil anaesthesia. Twenty-four hours after anaesthesia, no ectasia was observed. No histopathological changes were demonstrated in other tissues following anaesthesia. Results of the examinations suggest that the use of clove oil at a concentration of 30 mg·l -1 does not cause irreversible damage in European catfish.Acute toxicity, haematological profile, biochemical profile of blood, histological examination of tissues

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“…Evaluations were made in four consecutive phases (Thienpoint and Niemegeers, 1965;Yoshikawa et al, 1988): 1. acceleration and subsequent deceleration of opercular movements, a partial loss of reactivity to external stimuli 2. loss of equilibrium, opercular movements very slow, fish still reactive to strong stimuli 3. total loss of reactivity, fish are lying at the tank bottom and do not respond to handling 4. complete cessation of opercular movements, fish die if left in the bath for too long Lethal concentration levels (LC50, LC0.1 and LC99.9) were determined by the probit analysis using EKO-TOX 5.1 software.…”

Section: Acute Toxicity Of Clove Oilmentioning

confidence: 99%

Effects of clove oil anaesthesia on common carp (Cyprinus carpio L.)

Velíšek¹,

Svobodová²,

Piačková³

et al. 2005

Vet. Med. - Czech

107

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ABSTRACT:The aim of the study was to investigate acute toxicity of clove oil for common carp and, using values of haematological and biochemical profiles of blood and histological tissue examinations, to assess the effects of the fish exposure to that anaesthetic. Acute toxicity values of clove oil for carp were found 10 minLC50 74.3 mg/l; 10minLC0.1 51.6 mg/l; 10minLC99.9 110.1 mg/l; 96hLC50 18.10 mg/l; 96hLC0.1 15.45 mg/l; and 96hLC99.9 19.80 mg/l. The fish were divided into four groups for haematological and biochemical examinations of blood and histological examinations of tissues. The groups were Control I (before the anaesthetic administration), Experiment I (immediately after 10 min anaesthesia at the concentration of 30 mg/l), Experiment II (24 hrs after 10 min anaesthesia) and Control II (controls examined in parallel with Experiment II). A total of 40 carp were examined. Clove oil anaesthesia had not effect on the haematological profile. The 10-min exposure to clove oil at a concentration of 30 mg/l caused a significant (P < 0.01) increase in the concentration of glucose (GLU) and inorganic phosphate (PHOS) immediately after anaesthesia. Clove oil anaesthesia had not effect on other biochemical indices. Histological examination showed capillary ectasia of gill filaments immediately after clove oil anaesthesia. Twenty-four hours after anaesthesia, no ectasia was observed. No histopathological changes were demonstrated in other tissues following anaesthesia. Results of the examinations suggest that the use of clove oil at a concentration of 30 mg/l does not cause irreversible damage in carp.

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Anesthetic effect of CO2 on fish. I. Changes in depth of anesthesia of the carp anesthetized with a constant level of CO2.

Cited by 33 publications

References 0 publications

Effects of 2-phenoxyethanol anaesthesia on sheatfish (Silurus glanis L.)

Effects of 2-phenoxyethanol anaesthesia on sheatfish (Silurus glanis L.)

Effects of Clove Oil Anaesthesia on European Catfish (Silurus glanis L.)

Effects of clove oil anaesthesia on common carp (Cyprinus carpio L.)

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