Aneurysmal portahepatic venous fistula: report of two cases.

Chagnon, S; Vallée, C.; Barge, J; Chevalier, L J; Gál, Judit; Bléry, M

doi:10.1148/radiology.159.3.3517953

Cited by 108 publications

(43 citation statements)

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“…To date, there is no report of serious complication such as rupture or thrombosis, and prognosis is good. Operation has been reported in only one case in which encephalopathy could not be controlled by conservative means (4). The present case with no serious complication or no complaints did not require surgical treatment and will continue to be follow-up by US studies.…”

Section: Discussionmentioning

confidence: 55%

“…One embryologic explanation for shunts in the right lobe is the persistence of high flow communication between the omphalomesenteric venous system and the right horn of the sinus venosus (10). Decreasing tolerance toward toxic metabolites with age may possibly explain its late clinical manifestation (4,10). In the present case, the portal aneurysm may have preceded the venous shunt followed by rupture into the hepatic vein to establish the communication.…”

Section: Discussionmentioning

confidence: 66%

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Portal Hepatic Venous Shunt via an Intrahepatic Portal Vein Aneurysm.

et al. 1992

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A rare case of portal hepatic venous shunt (PHVS) via an intrahepatic portal vein aneurysm (PVA) is presented. A 66-year-old man was admitted for examination of a mass in the liver. Ultrasonography demonstrated a cystic lesion (15 mm in diameter) at the posterior superior segment of the right hepatic lobe which communicated with the right portal vein (RPV) and right hepatic vein (RHV). Superior mesenteric portography showed a biloculate aneurysm in RPV and PHVS. Color doppler ultrasonography indicated that flow in the tracts entered the aneurysmal cavity from RPV and drained into RHV. (Internal Medicine 31: 899-903, 1992)

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Section: Discussionmentioning

confidence: 55%

Section: Discussionmentioning

confidence: 66%

Portal Hepatic Venous Shunt via an Intrahepatic Portal Vein Aneurysm.

et al. 1992

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“…Patent DV is considered an intrahepatic type of portosystemic shunt, that connects the portal vein at its umbilical portion and the junction of the left hepatic vein and inferior vena cava. In the literature, the intrahepatic type of portovenous shunt between the left portal vein and distal part of left hepatic vein has sometimes been confused with the patent DV 17,18) . Anomalous shunt between the main portal vein and inferior vena cava can also exist, with the course of the shunt along the fissure for the ligamentum venosus 19,20) .…”

Section: Discussionmentioning

confidence: 99%

Transvenous Embolization of Patent Ductus Venosus in Two Adult Cases

2009

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Although the patent ductus venosus is congenital, the symptoms due to hyperammonemia vary, possibly depending on the shunt ratio and individual tolerance to ammonia. We have experienced two adult patients with large caliber patent ductus venosus and compensated liver cirrhosis. In one case, the ductus venosus simply communicated to the portal vein, but in the other case there were multiple short communicating vessels between the ductus venosus and left portal vein. Both cases were successfully treated by the transvenous embolization with steel coils and ethanolamine oleate, which has an expansive nature.

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“…Congenital portosystemic shunts are rare anomalies that can be seen incidentally when an infant undergoes ultrasonography (US) for other reasons. Several forms of intrahepatic portosystemic shunts have been reported (8,9). The most common form is a single large tube that connects the right PV to the inferior vena cava.…”

Section: Intrahepatic Vascular Shuntsmentioning

confidence: 99%

Cross-sectional imaging of congenital and acquired abnormalities of the portal venous system

Ozbayrak

Tatlı²

2016

Diagn Interv Radiol

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A wide range of congenital and acquired abnormalities may affect the portal venous system. They are sometimes encountered incidentally because of the widespread use of cross-sectional imaging modalities. Awareness of the presence of these abnormalities is important for adequate planning of surgical and percutaneous interventional procedures. The article reviews the normal anatomy, variations, and acquired pathologies involving the portal venous system as showed on computed tomography (CT) and magnetic resonance imaging (MRI). AnatomyThe portal venous system refers to the veins draining the gastrointestinal tract and spleen to the liver, excluding the inferior rectum and anal canal. The superior mesenteric vein (SMV) and splenic vein (SV) behind the neck of the pancreas form the main portal vein (PV) (Fig. 1). The inferior mesenteric vein (IMV) may drain into either the SV or into the confluence directly. The PV divides into the right and left PV. The right PV divides into anterior and posterior branches that supply the right liver lobe. The left PV courses horizontally and branches out to supply the lateral and medial segments of the left lobe (1,2). Congenital abnormalities Variations of the portal veinAnatomic variants of the branching pattern of the intrahepatic PV are present in approximately 20% of the population (1). The most common variant is trifurcation of the PV; the right PV does not exist and the PV divides into the right anterior, right posterior, and left branches (Fig. 2). Right anterior segmental branch arising from the left PV is another common variant (Fig. 3). Less common PV variations have been reported but their incidence has not been found to be higher than 2% (2). Awareness of these variants is crucial for planning surgery; for example, in a patient with right anterior segmental branch arising from the left PV, ligation of the left PV during left hepatectomy may lead to unexpected infarction of the anterior segment of the right liver lobe. Congenital agenesis of the portal veinCongenital agenesis of the PV is a rare malformation characterized by the absence of the PV and anomalous drainage of SMV and SV into the systemic circulation (Fig. 4). The PV is normally formed in 4-10 weeks of embryonic development via involution of the periintestinal vitelline venous loop. Atypical involution may cause a prebiliary, preduodenal, or duplicated PV, and excessive involution may result in the absence of the PV (3). Liver morphology is generally preserved. Congenital agenesis of the major branches of the PV is ABSTRACTKnowing the normal anatomy, variations, congenital and acquired pathologies of the portal venous system are important, especially when planning liver surgery and percutaneous interventional procedures. The portal venous system pathologies can be congenital such as agenesis of portal vein (PV) or can be involved by other hepatic disorders such as cirrhosis and malignancies. In this article, we present normal anatomy, variations, and acquired pathologies involving the portal venous system ...

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Aneurysmal portahepatic venous fistula: report of two cases.

Cited by 108 publications

References 0 publications

Portal Hepatic Venous Shunt via an Intrahepatic Portal Vein Aneurysm.

Portal Hepatic Venous Shunt via an Intrahepatic Portal Vein Aneurysm.

Transvenous Embolization of Patent Ductus Venosus in Two Adult Cases

Cross-sectional imaging of congenital and acquired abnormalities of the portal venous system

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