Angle-Closure Glaucoma

Progression in Primary Angle Closure Eyes

“…Other eyes after PAC may not progress to either a raised IOP or glaucomatous neuropathy. [1][2][3][4][5][6][7][8][9] To the best of our knowledge, there is no prospective study of the long term course of PAC eyes, or of predictive factors for progression in this subtype of PAC.…”

“…PAC or subacute angle closure has a distinct clinical and anatomic profile as compared with acute and chronic angle-closure glaucoma. [1][2][3][4][5][6][7][8][9] PAC or subacute angle closure eyes show evidence of angle closure in an occludable angle, without glaucomatous optic neuropathy. Therefore, such eyes could be expected to have a good long-term prognosis after laser iridotomy, if the mechanism of the angle-closure is pupillary block.…”

Pupillary Block and Angle-Closure Glaucoma

“…Figure 4 a, b and c illustrate schematically the changes in size and shape of the bulge in their dependence on the size of the pupil and hence the degree of resistance to the passage of aqueous humor through the pupil. The existence of a pupillary block mechanism as responsible for the angle closure was already claimed by Curran [2] and since then con firmed by Barkan [3], Chandler [4], Chandler and T rotter [5], Sugar [6] and many others. Its presence was, however, deduced from changes in the configuration of the chamber angle as seen gonioscopically before and after angle closure, and after the establishment of a bypass.…”

Prognosis in spontaneous phakolytic glaucoma.

“…Most workers (Irvine and Irvine, I952; Irvine, I957; Chandler, 1958;Goldberg, 1967;Duke-Elder, I969) are of the opinion that lens extraction is the only feasible means of eliminating the anterior chamber reaction and the rise in tension. Lowering the intraocular pressure by the use of acetazolamide and osmotic pressure agents (Irvine and Irvine, I952; Chandler, I958; Goldberg, I967; Duke-Elder, I969) and control of the anterior chamber reaction by steroids (Becker and Schaffer, I965) is recommended.…”