Anti-cardiolipin Antibody Associated with Acute Hemorrhagic Pancreatitis

Chang, K K; Kuo, Yow-Chii; Chiu, Cheug-Tang; Wu, Shyi-Shane; Huang, Chun‐Yen; Wu, Cheng‐Shyong; Liaw, Shu-Chen; Ho, Huei-Huang

doi:10.1097/00006676-199309000-00022

Cited by 10 publications

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“…As mentioned above, the abdominal pain in APS is an important initial presentation; it may also be due to a pancreatic involvement by microangiopathy 95. Recently such involvement has been reported in CAPS as well.…”

Section: Aps and Vaso-ischemic (Occlusive) Diseases With Neuropsychiamentioning

confidence: 91%

Antiphospholipid Syndrome and Vascular Ischemic (Occlusive) Diseases: An Overview

Atanassova

2007

Yonsei Med J

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Antiphospholipid syndrome (APS) is primarily considered to be an autoimmune pathological condition that is also referred to as "Hughes syndrome". It is characterized by arterial and/or venous thrombosis and pregnancy pathologies in the presence of anticardiolipin antibodies and/or lupus anticoagulant. APS can occur either as a primary disease or secondary to a connective tissue disorder, most frequently systemic lupus erythematosus (SLE). Damage to the nervous system is one of the most prominent clinical constellations of sequelae in APS and includes (i) arterial/venous thrombotic events, (ii) psychiatric features and (iii) other non-thrombotic neurological syndromes. In this overview we compare the most important vascular ischemic (occlusive) disturbances (VIOD) with neuro-psychiatric symptomatics, together with complete, updated classifications and hypotheses for the etio-pathogenesis of APS with underlying clinical and laboratory criteria for optimal diagnosis and disease management.

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Section: Aps and Vaso-ischemic (Occlusive) Diseases With Neuropsychiamentioning

confidence: 91%

Antiphospholipid Syndrome and Vascular Ischemic (Occlusive) Diseases: An Overview

Atanassova

2007

Yonsei Med J

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“…Multiple case reports exist of patients with pancreatitis and antiphospholipid syndrome [68][69][70][71][72]. In a case series of four patients, an association of pancreatitis with high levels of anticardiolipin was found [73].…”

Section: Pancreatic Involvement In Apsmentioning

confidence: 99%

Review of Endocrine Manifestations of Antiphospholid Syndrome

Petri¹

2010

CRR

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Endocrine manifestations of the antiphospholipid syndrome (APS) are very rare. Adrenal insufficiency, surprisingly usually due to venous infarction, is the most common. It can even be the presenting manifestation of APS, occur in patients with transient antiphospholipid antibodies or even in a neonate through transplacental transfer of maternal antiphospholipid antibodies. The presentation, differential diagnosis, and treatment of adrenal involvement due to APS will be reviewed. Other even rarer endocrine manifestations will also be briefly discussed.

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“…Abdominal pain in patients with the APS may be caused by pancreatic involvement by the microangiopathy that is characteristic of the aPL. The presentation may be acute, with abdominal pain and vomiting (195,196). Pancreatic involvement was noted in 6 of the 50 patients with catastrophic APS recently reviewed.…”

Section: Pancreatitismentioning

confidence: 99%

Unusual Manifestations of the Antiphospholipid Syndrome

Asherson

Cervera

2003

CRIAI

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The classical clinical picture of the antiphospholipid syndrome (APS) is characterized by venous and arterial thromboses, fetal losses and thrombocytopenia, in the presence of antiphospholipid antibodies (aPL), namely lupus anticoagulant (LA), anticardiolipin antibodies (aCL), or antibodies to the protein "cofactor" b2 glycoprotein I. Single vessel involvement or multiple vascular occlusions may give rise to a wide variety of presentations. Any combination of vascular occlusive events may occur in the same individual and the time interval between them also varies considerably from weeks to months or even years. Deep vein thrombosis, sometimes accompanied by pulmonary embolism, is the most frequently reported manifestation in this syndrome. Cerebrovascular accidents-either stroke or transient ischemic attacks-are the most common arterial thrombotic manifestations. Early and late fetal losses, premature births and pre-eclampsia are the most frequent fetal and obstetric manifestations. Additionally, several other clinical features are relatively common in these patients, i.e., thrombocytopenia, livedo reticularis, heart valve lesions, hemolytic anemia, epilepsy, myocardial infarction, leg ulcers, and amaurosis fugax. However, a large variety of other clinical manifestations have been less frequently described in patients with the APS, with prevalences lower than 5%. These include, among others, large peripheral or aortic artery occlusions, Sneddon's syndrome, chorea, transverse myelopathy, intracardiac thrombus, adult respiratory distress syndrome, renal thrombotic microangiopathy, Addison's syndrome, Budd-Chiari syndrome, nodular regenerative hyperplasia of the liver, avascular necrosis of the bone, cutaneous necrosis or subungual splinter hemorrhages. In this article, some of these "unusual" manifestations are reviewed.

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Anti-cardiolipin Antibody Associated with Acute Hemorrhagic Pancreatitis

Cited by 10 publications

References 0 publications

Antiphospholipid Syndrome and Vascular Ischemic (Occlusive) Diseases: An Overview

Antiphospholipid Syndrome and Vascular Ischemic (Occlusive) Diseases: An Overview

Review of Endocrine Manifestations of Antiphospholid Syndrome

Unusual Manifestations of the Antiphospholipid Syndrome

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