1993
DOI: 10.1001/archneur.1993.00540120016006
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Anti-Peripheral Nerve Myelin Antibodies and Terminal Activation Products of Complement in Serum of Patients With Acute Brachial Plexus Neuropathy

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Cited by 54 publications
(36 citation statements)
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“…Some studies have reported serum anti-ganglioside antibodies in NA patients, however it is still unclear whether these are actually involved in the pathogenesis of NA (3,21). In particular, the anti-GalNAc-GD1a antibody was reported as a candidate biological marker for predicting response to immune therapy (14).…”
Section: Discussionmentioning
confidence: 99%
“…Some studies have reported serum anti-ganglioside antibodies in NA patients, however it is still unclear whether these are actually involved in the pathogenesis of NA (3,21). In particular, the anti-GalNAc-GD1a antibody was reported as a candidate biological marker for predicting response to immune therapy (14).…”
Section: Discussionmentioning
confidence: 99%
“…Idiopathic LSP may be the lower extremity counterpart to idiopathic brachial neuritis. 5,10,15 Patients with idiopathic brachial neuritis may show complement-fixing antimyelin antibodies 17 and mononuclear inflammatory cell infiltrates of the affected plexus.…”
Section: Discussionmentioning
confidence: 99%
“…Suarez and colleagues showed the presence of mononuclear inflammatory infiltrates in brachial plexus biopsy specimens of patients with neuralgic amyotrophy 4 ; another study showed an increase in complement-fixing antibodies to peripheral nerve myelin in the acute phase of the condition in 3 patients. 5 A number of antecedent events that may serve as triggers for an immune-mediated etiology of neuralgic amyotrophy have been reported. These include viral infections, immunizations, and strenuous exercise, as well as the perioperative and peripartum periods.…”
Section: Etiologymentioning
confidence: 99%