2022
DOI: 10.1016/j.amsu.2022.104154
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Anti-tuberculosis drug-induced liver injury in patient with hepatitis B and cirrhosis: A case report

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Cited by 4 publications
(2 citation statements)
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“…The function of PPAR-γ activation against mycobacterial infection was shown to positively regulate prostaglandin (PG) E2 production in infected macrophages (23). Thus, PPAR-γ activation will increase cyclooxygenase (COX2) expression and PGE2 production in macrophages infected with M. tuberculosis so that macrophages will become activated macrophages and can kill M. tuberculosis more strongly (24). When macrophages can kill bacteria, the infection will gradually improve, and gamma interferon levels will decrease (25,26).…”
Section: Discussionmentioning
confidence: 99%
“…The function of PPAR-γ activation against mycobacterial infection was shown to positively regulate prostaglandin (PG) E2 production in infected macrophages (23). Thus, PPAR-γ activation will increase cyclooxygenase (COX2) expression and PGE2 production in macrophages infected with M. tuberculosis so that macrophages will become activated macrophages and can kill M. tuberculosis more strongly (24). When macrophages can kill bacteria, the infection will gradually improve, and gamma interferon levels will decrease (25,26).…”
Section: Discussionmentioning
confidence: 99%
“…The prevalence of cardiac cirrhosis is very difficult to identify because the majority of it is asymptomatic, where an increase in right atrial pressure is transmitted to the hepatic vein, causing congestion and an increase in sinusoidal pressure [ 2 ]. Advanced liver disease correlates with the risk of postoperative mortality [ [3] , [4] , [5] ]. The presence of portal hypertension, collateral circulation, and thrombocytopenia increases the risk of bleeding during and after surgery [ 6 ].…”
Section: Introductionmentioning
confidence: 99%