“…The idea of dissociation within the emotion schemas can be applied to alexithymia; ‘without linkages to images and words, the individual is unable to symbolize emotional states’ (Taylor & Bagby, ; p. 105). Moreover, activation of the subsymbolic system without symbolic connections may result in poorly regulated states of emotional arousal, which may contribute to the pathogenesis of different disorders, both somatic (Chatzi et al ., ; Conrad, Schilling, Langenbuch, Haidl, & Liedtke, ; Korkoliakou et al ., ; Martínez et al ., ; Vanheule, Vandenbergen, Verhaeghe, & Desmet, ; Verissimo, , ) and psychiatric (Carano et al ., ; Caretti & Craparo, ; Carpenter & Chung, ; De Panfilis, Ossola, Tonna, Catania, & Marchesi, ; Kessler, Schwarze, Filipic, Traue, & Von Wietersheim, ; Li, Zhang, Guo, & Zhang, ; Ogrodniczuk, Sochting, Piper, & Joyce, ; Parker, Wood, Bond, & Shaughnessy, ; Pinaquy, Chabrol, Simon, Louvet, & Barbe, ; Rutten et al ., ), with which alexithymia has been associated (Solano, ; Taylor, ; Taylor & Bagby, ). However, the literature also indicates some rare studies that showed no differences between the scores of alexithymia obtained by clinical and general population measured with the TAS‐20 (Vinai et al ., ) or a non‐significant association between alexithymic characteristics measured with the TAS‐20 and a somatic or psychiatric disorder (Barbasio et al ., ; Kooiman, Bolk, Brand, Trijsburg, & Rooijmans, ).…”