“…It is well established that arterial wall biomechanics (ie, wall motion, wall stress, and wall thickness) contribute to the localization of atherosclerotic plaques and aneurysm development/progression. [1][2][3][4][5] Hemodynamic factors have been linked to the localization of atherosclerotic plaque formation in areas of low shear stress and shear separation in the carotid, aortic, femoral, and coronary arteries. 6 Furthermore, the elastic properties of the aortic wall permit deformation of the aorta with pulsatile blood flow: ie, the aorta relaxes during diastole and expands during systole.…”