Aphasic seizure caused by focal epilepsy in the left fusiform gyrus

Suzuki, I; Shimizu, Hiroyuki; Ishijima, Buichi; Tani, Kazunori; Sugishita, Morihiro; Adachi, Naoto

doi:10.1212/wnl.42.11.2207

Cited by 28 publications

(11 citation statements)

References 0 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…The long duration of the dysphasia, approximately a few weeks, was compatible with status epilepticus, and PLEDshave been recognized to represent status epilepticus on electroencephalograph (6). Epileptic dysphasia is a rare clinical condition (7)(8)(9)(10)(11)(12)(13)(14)(15)(16)(17)(18)(19), and the present case is noteworthy because of the prolonged dysphasia with persistent PLEDswhich persisted throughout the recording time. Although Wells CR (19) reported a case of epileptic dysphasia and PLEDsappeared transiently in that case, noneof the previous cases of epileptic dysphasia were accompanied by persistent PLEDsas the present case.…”

Section: Discussionmentioning

confidence: 57%

Dysphasia Accompanied by Periodic Lateralized Epileptiform Discharges.

et al. 1997

View full text Add to dashboard Cite

Weencountered a 67-year-old man who presented with repetitious dysphasia accompanied by periodic lateralized epileptiform discharges (PLEDs) on the electroencephalogram.A good correlation was established between the dysphasia and the PLEDs.A persistent partial seizure accompanied by PLEDs originating in the left hemisphere presented with dysphasia clinically. None of the previously reported patients with epileptic dysphasia had accompanying PLEDs. The administration of carbamazepinewas successful to terminate the seizure, howevervalproate was not. This case indicates that PLEDsmay produce significant, however reversible, functional damage, and may advance our understanding of the pathophysiology of epileptic dysphasia. (Internal Medicine 36: 59-61, 1997)

show abstract

Section: Discussionmentioning

confidence: 57%

Dysphasia Accompanied by Periodic Lateralized Epileptiform Discharges.

et al. 1997

View full text Add to dashboard Cite

show abstract

“…This relationship was quite consistent across participants. Dense anatomical connections are known to exist between basal temporal and temporoparietal areas (Luders et al, 1986;Suzuki, Shimizu, & Tani, 1992), and the relative timing observed between these two areas may depend on these connections. Activity in medial temporal areas was concurrent with that in temporoparietal areas, and careful inspection of the data suggested either a coincident or phasic relationship between activity in the two areas.…”

Section: Discussionmentioning

confidence: 99%

Relative Timing of Neuronal Activity in Distinct Temporal Lobe Areas During a Recognition Memory Task for Words

Breier¹,

Simos²,

Zouridakis³

et al. 1998

Journal of Clinical and Experimental Neuropsychology

View full text Add to dashboard Cite

Nine neurologically normal volunteers engaged in a recognition memory task for visually presented words while the magnetic flux normal to the scalp surface was measured with a whole-head neuromagnetometer. The relative timing of activity occurring during the task in different brain areas was determined. Occipital lobe areas became active first, followed by basal temporal areas. Temporoparietal and medial temporal areas became active next, during later portions of the recorded epoch, and appeared to activate in concert. Except for the occipital lobe, activity was predominantly in the left hemisphere. This pattern was consistent across participants. Results suggest that MEG, in capturing temporal as well as spatial aspects of brain activity, has a promising role as an adjunct to other functional imaging modalities.

show abstract

“…As an ictal phenomenon (during which the patient is alert but dysphasic) there is usually a correlation between the clinical seizure and simultaneous EEG changes, denoting corresponding electrical ictal activity that would cease once the clinical fit was over [9,10]. In our patient, however, such a pattern was not seen.…”

Section: Discussionmentioning

confidence: 57%

“…In relation to epilepsy, aphasia can be either a manifestation of an ictal as in partial complex seizures or a post-ictal phenomenon (todd phenomenon) [3,[9][10][11]. As an ictal phenomenon (during which the patient is alert but dysphasic) there is usually a correlation between the clinical seizure and simultaneous EEG changes, denoting corresponding electrical ictal activity that would cease once the clinical fit was over [9,10].…”

Section: Discussionmentioning

confidence: 99%

Recurrent Seizures and Prolonged Post-Ictal Aphasia in a Patient with Multiple Sclerosis

Al-Shubaili

Farah

Khuraibet³

et al. 1998

Med Princ Pract

View full text Add to dashboard Cite

Objective: To evaluate the duration and underlying pathology of post-ictal aphasia in a patient with multiple sclerosis. Clinical Presentation: A 36-year-old female patient with chronic multiple sclerosis developed recurrent seizures followed by prolonged stereotyped post-ictal aphasia that lasted for 4–6 days. There was no response to a therapeutic trial of steroids. Investigations: A brain MRI did not reveal new demyelinative plaques. EEG showed persistent abnormalities from the left temporal lobe, which had no temporal relationship to the aphasic episodes. Conclusion: We believe that these aphasic episodes represent a post-ictal phenomenon of unusually long duration that has not been reported before.

show abstract

Aphasic seizure caused by focal epilepsy in the left fusiform gyrus

Cited by 28 publications

References 0 publications

Dysphasia Accompanied by Periodic Lateralized Epileptiform Discharges.

Dysphasia Accompanied by Periodic Lateralized Epileptiform Discharges.

Relative Timing of Neuronal Activity in Distinct Temporal Lobe Areas During a Recognition Memory Task for Words

Recurrent Seizures and Prolonged Post-Ictal Aphasia in a Patient with Multiple Sclerosis

Contact Info

Product

Resources

About