Apraxia in deep cerebral lesions.

Agostoni, Elio; Coletti, A.; Orlando, G.; Tredici, G

doi:10.1136/jnnp.46.9.804

Cited by 63 publications

(21 citation statements)

References 14 publications

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“…Several adults with thalamic lesions and apraxia have been reported (Cappa and Vignolo 1979, Alexander and LoVerme 1980, Agostoni et al 1983, Graff-Radford et al 1984, DeRenzi et al 1986, Della Salla et al 1992, Shuren et al 1994, most in association with an aphasia. Nadeau et al (1994) suggest that the praxis deficit seen with thalamic lesions is a reflection of a declarative memory deficit.…”

Section: Discussionmentioning

confidence: 99%

Acquired aphasia in children after surgical resection of left‐thalamic tumors

Nass¹,

Boyce²,

Leventhal³

et al. 2000

Develop Med Child Neuro

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Five children (three males, two females; four right‐, one left‐handed; age range 6 to 14 years) who developed aphasia after gross‐total excision of left predominantly thalamic tumors are reported. Three patients had Broca aphasia, one had mixed transcortical aphasia, and one patient had conduction aphasia. In the months after surgery, three children improved while receiving radiation and/or chemotherapy, although none recovered completely. Two patients with malignant tumors developed worsening aphasia when the tumor recurred, and later died. Two of three patients tested had visuospatial difficulties in addition to language deficits. Attention and executive functioning were affected in three of three patients tested. Memory, verbal and/or visual functioning, were affected in four of four patients tested. Both patients who were tested showed transient right hemineglect. Two of two patients tested were probably apraxic. The wide range of deficits in these children highlights the importance of the thalamus and other subcortical structures in developing cognition.

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Section: Discussionmentioning

confidence: 99%

Acquired aphasia in children after surgical resection of left‐thalamic tumors

Nass¹,

Boyce²,

Leventhal³

et al. 2000

Develop Med Child Neuro

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“…In 1983 Agostoni et al published seven cases of patients with lesions of the basal ganglia developing ideomotor limb apraxia, more studies on apraxia in patients with subcortical lesions were reported soon thereafter [1,32]. Moreover, in 1991 Shelton [43] found ideomotor apraxia in three of nine patients with Huntington's disease (HD), a rare hereditary,devastating neurodegenerative disease.…”

Section: Introductionmentioning

confidence: 97%

Ideomotor limb apraxia in Huntington's disease

Hödl

Otti

et al. 2008

J Neurol

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“…The first cases of ideomotor limb apraxia in patients with diseases that were subcortical in origin were reported in the 1980s, when case reports were published on apraxic patients with vascular subcortical dementia, PD and normal-pressure hydrocephalus [13,14] . Similarly, apraxia in HD, where predominantly subcortical structures are damaged, was first identified by Shelton in 1991, after which 2 further similar studies followed [15][16][17] .…”

Section: Introductionmentioning

confidence: 99%

Impaired Ideomotor Limb Apraxia in Cortical and Subcortical Dementia: A Comparison of Alzheimer’s and Huntington’s Disease

Holl¹,

Ille

Wilkinson

et al. 2011

Neurodegener Dis

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Background: Although ideomotor limb apraxia is often considered to occur only in dementia with cortical involvement like Alzheimer’s disease (AD), it is also frequently seen in dementia with subcortical degeneration like Huntington’s disease (HD). Methods: To assess the occurrence of ideomotor limb apraxia, 46 patients with HD (27 men) and 37 patients with AD (16 men), matched for cognitive performance, were assessed with an apraxia test battery containing tests of the imitation of meaningless hand and finger gestures, the performance of meaningful gestures and of pantomimic movements. Results: There was a high frequency of ideomotor limb apraxia in both AD and HD patients. For the assessment of hands’ imitation 13.5% of the AD patients and 41.3% of the HD patients were apraxic, for fingers’ imitation 21.6% (AD) and 41.3% (HD) were apraxic, for gestures 27.0% (AD) and 32.6% (HD), and for the assessment of pantomimic movements 24.3% (AD) and 52.2% (HD) showed apraxia. In the AD patients, disease severity was related to the occurrence of apraxia. Conclusions: Ideomotor limb apraxia is a common sign in both groups of patients, occurring in a high percentage. For particular neuropsychological deficits, including ideomotor limb apraxia, a division of dementia in a subcortical and cortical subtype seems to be clinically not meaningful.

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Apraxia in deep cerebral lesions.

Cited by 63 publications

References 14 publications

Acquired aphasia in children after surgical resection of left‐thalamic tumors

Acquired aphasia in children after surgical resection of left‐thalamic tumors

Ideomotor limb apraxia in Huntington's disease

Impaired Ideomotor Limb Apraxia in Cortical and Subcortical Dementia: A Comparison of Alzheimer’s and Huntington’s Disease

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