Apraxia of lid opening is alleviated by pallidal stimulation in a patient with Parkinson’s disease

Goto, Satoshi; Kihara, Kazunori; Hamasaki, Tadashi; Nishikawa, Sadakatsu; Hirata, Yusuke; Ushio, Yukitaka

doi:10.1046/j.1468-1331.2000.00058.x

Cited by 22 publications

(13 citation statements)

References 10 publications

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“…48,50,83,84,88,90,91 In one patient, apraxia of eyelid opening was improved by right GPi DBS. 92 In bilateral stimulation, OFF-period motor UPDRS score was improved by 40 to 50%, 50,82,84,91 and benefit on gait and posture was clear. 93 Patients with unilateral GPi stimulation experienced a general improvement in selfreported quality of life that exceeded the purely motor and physical aspects of quality of life.…”

Section: Results Of Gpi Dbsmentioning

confidence: 96%

Treatment results: Parkinson's disease

et al. 2002

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Deep brain stimulation (DBS) is a neurosurgical treatment of Parkinson's disease that is applied to three targets: the ventral intermediate nucleus of the thalamus (Vim), the globus pallidus internas (GPi) and the subthalamic nucleus (STN). Vim DBS mainly improves contralateral tremor and, therefore, is being supplanted by DBS of the two other targets, even in patients with tremor dominant disease. STN and GPi DBS improve off-motor phases and dyskinesias. There is little comparative data between these procedures. The magnitude of the motor improvement seems more constant with STN than GPi DBS. STN DBS allows a decrease in antiparkinsonian drug doses and consumes moderate current. These advantages of STN over GPi DBS are offset by the need for more intensive postoperative management. The DBS procedure has the unique advantage of reversibility and adjustability over time. Patients with young-onset Parkinson's disease suffering from levodopa-induced motor complications but still responding well to levodopa and who exhibit no behavioral, mood, or cognitive impairment benefit the most from STN DBS. Adverse effects more specific of the DBS procedure are infection, cutaneous erosion, and lead breaking or disconnection. Intracranial electrode implantation can induce a hematoma or contusion. Most authors agree that the benefit to risk ratio of DBS is favorable.

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Section: Results Of Gpi Dbsmentioning

confidence: 96%

Treatment results: Parkinson's disease

et al. 2002

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“…Treatment is challenging and consists of reducing voltage, increasing frequency, and administering levodopa in addition to conventional therapies. Paradoxically, some patients with AEO experience improvement with STN (151, 152) or GPi (153) DBS.…”

Section: Apraxia Of Eyelid Opening and Closurementioning

confidence: 99%

Eyelid Dysfunction in Neurodegenerative, Neurogenetic, and Neurometabolic Disease

Hamedani

Gold

2017

Front. Neurol.

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Eye movement abnormalities are among the earliest clinical manifestations of inherited and acquired neurodegenerative diseases and play an integral role in their diagnosis. Eyelid movement is neuroanatomically linked to eye movement, and thus eyelid dysfunction can also be a distinguishing feature of neurodegenerative disease and complements eye movement abnormalities in helping us to understand their pathophysiology. In this review, we summarize the various eyelid abnormalities that can occur in neurodegenerative, neurogenetic, and neurometabolic diseases. We discuss eyelid disorders, such as ptosis, eyelid retraction, abnormal spontaneous and reflexive blinking, blepharospasm, and eyelid apraxia in the context of the neuroanatomic pathways that are affected. We also review the literature regarding the prevalence of eyelid abnormalities in different neurologic diseases as well as treatment strategies (Table 1).

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“…With the patient under general anesthesia with propofol, quadripolar deep brain stimulation (DBS) electrodes (Model 3387, Medtronic Inc., Minneapolis, MN) were implanted in the bilateral GPi (Fig. 2) with the aid of magnetic resonance imaging (MRI), third ventriculography, and microelectrode guidance 21–23. The optimal targets were determined to be 2 mm anterior and 20 mm lateral to the midpoint of the anterior to posterior commissure line, and 1 mm dorsal to the third ventricle floor, as previously described 21, 23.…”

Section: Case Reportmentioning

confidence: 99%

Bilateral pallidal stimulation for idiopathic segmental axial dystonia advanced from meige syndrome refractory to bilateral thalamotomy

et al. 2001

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Meige syndrome is an adult-onset dystonic movement disorder that predominantly involves facial muscles, while some patients with this syndrome develop spasmodic dysphonia and dystonia of the neck, trunk, arms, and legs. We report that all dystonic symptoms that had been refractory to both pharmacotherapy and bilateral thalamotomy were markedly alleviated by bilateral pallidal stimulation in a patient with segmental axial dystonia advanced from Meige syndrome.

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Apraxia of lid opening is alleviated by pallidal stimulation in a patient with Parkinson’s disease

Cited by 22 publications

References 10 publications

Treatment results: Parkinson's disease

Treatment results: Parkinson's disease

Eyelid Dysfunction in Neurodegenerative, Neurogenetic, and Neurometabolic Disease

Bilateral pallidal stimulation for idiopathic segmental axial dystonia advanced from meige syndrome refractory to bilateral thalamotomy

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