Arsenic removal of high-arsenic wastewater from gallium arsenide semiconductor production by enhanced two-stage treatment

Li, Anfeng; Luo, Jianping; Xu, Wenju; Pan, Tao

doi:10.1080/19443994.2014.927795

Cited by 6 publications

(3 citation statements)

References 23 publications

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“…As-product manufacturing will also contribute to contamination of soil and water bodies, i.e. contamination from a pesticide manufacturing facility have led to As aqueous concentrations in groundwater of up to 0.2 mM (O’day et al 2004); the wastewater effluent from a Gallium Arsenide semiconductor production facility was reported to have an As concentration of 0.8 mM (Li et al 2015). Exposure to high As levels have obligated many microorganisms to adapt to the inhibitory effects of As, and develop efficient detoxifying mechanisms ( i.e., precipitation, chelation, compartimentalization) (Slyemi and Bonnefoy 2012) and As extrusion (Cervantes et al 1994).…”

Section: Introductionmentioning

confidence: 99%

Adaptation of a Methanogenic Consortium to Arsenite Inhibition

Rodríguez-Freire

Moore

Sierra-Álvarez

et al. 2015

Water Air Soil Pollut

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Arsenic (As) is a ubiquitous metalloid known for its adverse effects to human health. Microorganisms are also impacted by As toxicity, including methanogenic archaea, which can affect the performance of process in which biological activity is required (i.e. stabilization of activated sludge in wastewater treatment plants). The novel ability of a mixed methanogenic granular sludge consortium to adapt to the inhibitory effect of arsenic (As) was investigated by exposing the culture to approximately 0.92 mM of AsIII for 160 d in an arsenate (AsV) reducing bioreactor using ethanol as the electron donor. The results of shaken batch bioassays indicated that the original, unexposed sludge was severely inhibited by arsenite (AsIII) as evidenced by the low 50% inhibition concentrations (IC50) determined, i.e., 19 and 90 μM for acetoclastic- and hydrogenotrophic methanogenesis, respectively. The tolerance of the acetoclastic and hydrogenotrophic methanogens in the sludge to AsIII increased 47-fold (IC50 = 910 μM) and 12-fold (IC50= 1100 μM), respectively, upon long-term exposure to As. In conclusion, the methanogenic community in the granular sludge demonstrated a considerable ability to adapt to the severe inhibitory effects of As after a prolonged exposure period.

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Section: Introductionmentioning

confidence: 99%

Adaptation of a Methanogenic Consortium to Arsenite Inhibition

Rodríguez-Freire

Moore

Sierra-Álvarez

et al. 2015

Water Air Soil Pollut

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“…As shown in Figure 2C, compared to manganese oxide alone (0.2 g L −1 ), As(III) oxidation by manganese oxide in the presence of PMS considerably increased to 95.9% after 30 min. The chemical oxidants of H 2 O 2 and NaClO, which had been investigated for As(III) oxidation in previous literature, were also employed to evaluate their As(III) oxidation activities for comparing with the manganese oxide/PMS system. After 30 min, the As(III) oxidation rates of H 2 O 2 and NaClO were 22.6% and 30.3%, respectively (Figure 2C), which were far lower than that of manganese oxide in the presence of PMS.…”

Section: Resultsmentioning

confidence: 99%

Peroxymonosulfate Improves the Activity and Stability of Manganese Oxide for Oxidation of Arsenite to Arsenate

Liu

Hou

Hartley

2020

CLEAN Soil Air Water

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Manganese oxide has been widely investigated for oxidation of arsenite (As(III)) to arsenate (As(V)) due to its high redox potential; however, it becomes extremely unstable after reuse. Here, As(III) oxidation activity and stability of manganese oxide in the presence of peroxymonosulfate (PMS) is investigated. Batch experimental results reveal that manganese oxide/PMS exhibits high catalytic activity for As(III) oxidation compared to manganese oxide or PMS alone. Addition of PMS to manganese oxide not only reveals long-term stability for As(III) oxidation, but also shows high As(III) oxidation activity in the presence of coexisting ions such as As(V) and phosphate. Quenching tests reveal that As(III) oxidation in the manganese oxide/PMS system is attributed to activation of PMS by manganese oxide at different oxidation states (Mn(III) and Mn(IV)), and the generation of sulfate radicals that are responsible for As(III) oxidation. exist in polluted groundwater. Between them, As(III) is reportedly more harmful than As(V) due to its greater mobility and toxicity. [4] Therefore, oxidation of As(III) to As(V) is not only a possible approach to reducing arsenic impacts on human health, but also is a vital step for total arsenic immobilization prior to treatment by subsequent technologies such as sorption and coprecipitation. [5] Manganese oxide has been widely used for rapid oxidation of As(III) to As(V) due to its high redox potential of E 0 (MnO 2 / Mn 2+ ) = 1.22 V. [6][7][8][9][10][11][12][13][14][15] However, to our knowledge, manganese oxide is unstable after being reused for As(III) oxidation, which is attributed to the release of Mn 2+ during As(III) oxidation and a decrease in the Mn oxidation state. [8,9] This means that manganese oxide only acts as an oxidant for As(III) oxidation, rather than as a catalyst. In addition, a reduction in oxidation activity may occur in the presence of coexisting ions such as phosphate in arsenic-containing wastewater. [14][15][16][17] This is due to the favorable adsorption of coexisting ions onto manganese oxide surfaces, which then occupy and passivate its surface active sites. [16] Therefore, a strategy to improve As(III) oxidation rate and guarantee the recycling of manganese oxide activity is still required.Peroxymonosulfate (PMS, HSO 5 − ) is one form of peroxosulfate-based chemical that has been widely used as an oxidant in different advanced oxidation process (AOP) techniques for degradation of organic pollutants, which has been used to quickly oxidize As(III) to As(V) in recent works. [18,19] Due to the low As(III) oxidation rate of PMS at room temperature, there is still a need to activate PMS forming sulfate radicals (SO 4 −• ) with high redox potential (2.5-2.8 V). In peroxosulfate-based AOP systems, sulfate radicals (SO 4 −• ) can be formed through activation of PMS by UV, heat, and catalysts. [20][21][22] For example, peroxosulfate-based chemicals activated by UV and Fe 2+ rapidly oxidized As(III). [18,23] As a result of lower energy consumption and its recycling ...

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“…Relatively high concentrations of As have been found in effluent streams of electronic fabrication facilities that utilize III/V materials in several countries ,000 mg/L). 9,12,13 High levels of arsenide particulates are also expected in CMP waste streams, 14,15 but the concentrations found in these effluents have not been reported to date. The CMP effluents are treated at semiconductor facilities to remove regulated pollutants (eg, copper, soluble As) prior to discharge to municipal sewer systems, but current treatment systems are not designed to remove nano-sized Ga-and In arsenides or other Ga-and In-containing NPs.…”

Section: Introductionmentioning

confidence: 99%

Cytotoxicity Assessment of Gallium- and Indium-Based Nanoparticles Toward Human Bronchial Epithelial Cells Using an Impedance-Based Real-Time Cell Analyzer

et al. 2020

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The semiconductor manufacturing sector plans to introduce III/V film structures (eg, gallium arsenide (GaAs), indium arsenide (InAs) onto silicon wafers due to their high electron mobility and low power consumption. Aqueous solutions generated during chemical and mechanical planarization of silicon wafers can contain a mixture of metal oxide nanoparticles (NPs) and soluble indium, gallium, and arsenic. In this work, the cytotoxicity induced by Ga- and In-based NPs (GaAs, InAs, Ga2O3, In2O3) and soluble III-V salts on human bronchial epithelial cells (16HBE14o-) was evaluated using a cell impedance real-time cell analysis (RTCA) system. The RTCA system provided inhibition data at different concentrations for multiple time points, for example, GaAs (25 mg/L) caused 60% inhibition after 8 hours of exposure and 100% growth inhibition after 24 hours. Direct testing of As(III) and As(V) demonstrated significant cytotoxicity with 50% growth inhibition concentrations after 16-hour exposure (IC50) of 2.4 and 4.5 mg/L, respectively. Cell signaling with rapid rise and decrease in signal was unique to arsenic cytotoxicity, a precursor of strong cytotoxicity over the longer term. In contrast with arsenic, soluble gallium(III) and indium(III) were less toxic. Whereas the oxide NPs caused low cytotoxicity, the arsenide compounds were highly inhibitory (IC50 of GaAs and InAs = 6.2 and 68 mg/L, respectively). Dissolution experiments over 7 days revealed that arsenic was fully leached from GaAs NPs, whereas only 10% of the arsenic was leached out of InAs NPs. These results indicate that the cytotoxicity of GaAs and InAs NPs is largely due to the dissolution of toxic arsenic species.

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Arsenic removal of high-arsenic wastewater from gallium arsenide semiconductor production by enhanced two-stage treatment

Cited by 6 publications

References 23 publications

Adaptation of a Methanogenic Consortium to Arsenite Inhibition

Adaptation of a Methanogenic Consortium to Arsenite Inhibition

Peroxymonosulfate Improves the Activity and Stability of Manganese Oxide for Oxidation of Arsenite to Arsenate

Cytotoxicity Assessment of Gallium- and Indium-Based Nanoparticles Toward Human Bronchial Epithelial Cells Using an Impedance-Based Real-Time Cell Analyzer

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