Artificial Respiration With the Tunnicliffe Breathing-Jacket

Spalding, J. M. K.; Opie, L.H.

doi:10.1016/s0140-6736(58)90869-9

Cited by 21 publications

(2 citation statements)

References 6 publications

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“…Improvement in breathlessness and exercise tolerance has been reported by all subjects. Although no formal assessment of either variable has been Before With attempted, 11 hospital for many weeks. A further 14% of with skeletal deformity had already retired t they presented.…”

Section: Resultsmentioning

confidence: 99%

Domiciliary ventilatory support: an analysis of outcome.

Sawicka

Loh

Branthwaite

1988

Thorax

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Section: Resultsmentioning

confidence: 99%

Domiciliary ventilatory support: an analysis of outcome.

Sawicka

Loh

Branthwaite

1988

Thorax

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“…It readily gained acceptance as a means of treating patients with poliomyelitis and other neuromuscular disorders. The description in 1958 of the Tunnicliffe breathing-jacket [2] contributed to the development of a variety of cuirass ventilators but the principle of the apparatus remained the same, that is a negative pressure cycle is applied outside the body whilst ambient pressure is maintained at the mouth.…”

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confidence: 99%

Acute effects of external negative pressure ventilation in chronic obstructive pulmonary disease compared with normal subjects

Cooper¹,

Harris²,

Howard³

1991

Eur Respir J

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This study compares the acute physiological effects of external negative pressure ventilation (ENPV) in normal subjects and patients with chronic obstructive pulmonary disease (COPD). The equipment consisted of an airtight jacket (Pneumosuit) and vacuum pump. Minute ventilation (Ve) was recorded using a light-emitting turbine transducer. Oxygen uptake (VO2) and carbon dioxide output (VCO2) were calculated every 30 s. Measurements were made at rest and during ENPV with pressures of -20 cmH2O and -40 cmH2O. The ventilator rate was fixed at 16.min-1. In 10 normal subjects, Ve increased from 8.6 to 22.9 l.min-1 (p less than 0.01) accompanied by an increase in VCO2 from 0.25 to 0.39 l.min-1 (p less than 0.01). In 10 normocapnic COPD patients (arterial carbon dioxide tension (PaCO2) less than 6.0 kPa) Ve increased from 11.5 to 17.1 l.min-1 (p less than 0.01) whilst in 10 hypercapnic patients (PaCO2 greater than 6.0 kPa) Ve increased from 9.7 to 12.4 l.min-1 (p less than 0.01). A change in VCO2 was not detected in the COPD patients, and VO2 did not change in any group. Arterial blood samples were obtained in eight hypercapnic patients. Baseline mean physiological deadspace ventilation (VD) was calculated to be 4.9 l.min-1 (56% of Ve) whilst Ve was 8.8 l.min-1 in this subgroup. During ENPV, arterial oxygen tension (PaO2) increased from 6.8 to 8.2 kPa (p less than 0.01) whilst PaCO2 decreased from 6.8 to 5.8 kPa (p less than 0.01) suggesting that despite the large physiological deadspace, a significant increase in alveolar ventilation had occurred. In advanced COPD, thoracic compliance falls and limits the ventilatory response to ENPV.(ABSTRACT TRUNCATED AT 250 WORDS)

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Sleep, Breathing, and Neurologic Disorders

Chokroverty

Provini

2017

Sleep Disorders Medicine

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Artificial Respiration With the Tunnicliffe Breathing-Jacket

Cited by 21 publications

References 6 publications

Domiciliary ventilatory support: an analysis of outcome.

Domiciliary ventilatory support: an analysis of outcome.

Acute effects of external negative pressure ventilation in chronic obstructive pulmonary disease compared with normal subjects

Sleep, Breathing, and Neurologic Disorders

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