Ascitic fluid ?1-antitrypsin

Villamil, Federico; Sorroche, Patricia; Aziz, H; López, Patricia; Oyhamburu, José

doi:10.1007/bf01537582

Cited by 15 publications

(6 citation statements)

References 19 publications

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“…74,75 These have often been looked at in combination with other parameters such as SAAG, LD and cholesterol and multivariate statistical methods used to improve diagnostic accuracy. 24,58,59,74 There are case reports of other tumour markers such as prostate-specific antigen. 76 Overall there appears to be little added value from measurement of the traditional tumour markers in ascitic fluid compared with measurement in serum and the same limitations with respect to using these for diagnosis apply.…”

Section: Amylase (Is Pancreatitis the Cause?)mentioning

confidence: 99%

“…These include well-established ‘tumour markers’ such as AFP, CEA, CA19-9 and CA-125, as well as inflammatory markers such as ferritin, caeruloplasmin, α -2 macroglobulin, α -1-anti-trypsin, interleukins and other proteins such as fibronectin and complement. 74,75 These have often been looked at in combination with other parameters such as SAAG, LD and cholesterol and multivariate statistical methods used to improve diagnostic accuracy. 24,58,59,74 There are case reports of other tumour markers such as prostate-specific antigen.…”

Section: Tests Used In the Biochemical Analysis Of Peritoneal Fluidsmentioning

confidence: 99%

Section: Tests Used In the Biochemical Analysis Of Peritoneal Fluidsmentioning

confidence: 99%

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Biochemical analysis of ascitic (peritoneal) fluid: what should we measure?

Tarn

Lapworth

2010

Ann Clin Biochem

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Ascitic fluid samples are frequently sent to the laboratory for analysis. Although the underlying cause of the ascites is often thought to be clinically obvious, it is important to establish a definitive diagnosis. The value of a cell count and bacterial culture of the ascitic fluid is not disputed, but the role of biochemical testing is less clear. The use of ascitic fluid total protein to try to classify ascitic fluids as either an exudate or a transudate has contributed to this. The use of the physiologically based serum ascites albumin gradient to differentiate ascites caused by portal hypertension from other causes provides a better diagnostic approach. We recommend that the serum ascites albumin gradient is performed by laboratories as the first-line test and that interpretative reports are provided. Additional testing should be restricted to specific diagnostic queries and requires close collaboration between the laboratory and the clinician.

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Section: Amylase (Is Pancreatitis the Cause?)mentioning

confidence: 99%

Section: Tests Used In the Biochemical Analysis Of Peritoneal Fluidsmentioning

confidence: 99%

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Biochemical analysis of ascitic (peritoneal) fluid: what should we measure?

Tarn

Lapworth

2010

Ann Clin Biochem

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“…In cases with a malignant genesis, the values were nearly always elevated to > 120 mg/dl. (64) 13. Other examinations focused on the biochemical differentiation of ascites, such as determining cholinesterase, coagulant activity (22) and interleukin-6 (18) as sensitive parameters for bacterial infection as well as evaluating the receptors (p55 and p75) for the tumour necrosis factor.…”

Section: Ferritinmentioning

confidence: 99%

Oedema and ascites

Hepatology Textbook and Atlas

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Water and electrolyte balance 294 2 Definition 295 2.1 States of hydration 295 2.2 Oedema and anasarca 295 2.3 Ascites 296 3 Pathogenesis 296 3.1 Oedematization 296 3.2 Formation of ascites 297 3.2.1 Mechanical factors 297 3.2.2 Biochemical factors 298 3.2.3 Increase in renal sodium retention 300 3.2.4 Theories of ascites formation 300 4 Aetiology of ascites 302 4.1 Differential diagnosis 302 4.2 Hepatogenic ascites 302 5 Diagnosis of ascites 303 5.1 Clinical findings 303 5.2 Imaging procedures 304 5.3 Laboratory diagnosis 305 6 Complications of ascites 308 7 Spontaneous bacterial peritonitis 308 7.1 Definition 3 0 8 7.2 Forms and frequency 308 7.3 Pathogenesis and predisposing factors 309 7.4 Clinical aspects 309 7.5 Prophylaxis and therapy 309 8 Conservative therapy of ascites 310 8.1 Prophylaxis 310 8.2 Basic therapy (stage I) 311 8.3 Diuretic therapy (stage II) 312 8.3.1 Pharmacology of diuretics 312 8.3.2 Side effects 314 8.3.3 Hyponatraemia 314 8.3.4 Resistance to diuretics 314 8.4 Osmotic diuresis (stage III) 314 8.5 Paracentesis (stage IV) 315 9 Refractory ascites 316 10 Invasive therapeutic procedures 316 10.1 Ascites reinfusion 316 10.2 Peritoneovenous shunt 317 10.3 TIPS 320 11 Surgical treatment 321 12 Liver transplantation 322 ț References (1Ϫ240) 323 (Figures 16.1Ϫ16.17; tables 16.1Ϫ16.19) 16 Oedema and ascites 1 Water and electrolyte balance ᭤ Water is an indispensable factor of life. By means of carefully coordinated regulatory mechanisms, the water equilibrium and hence the reservoir of body water is held constant. It is important to keep water intake and output in balance to maintain isovolaemia. (s. fig. 16.1) Water is present in a free (non-osmotically bound) state and as a chemically bound hydrate solid structure. • The clearance of freewater is controlled by vasopressin; it is calculated from the volume of urine/minute minus the osmolal clearance. A normal daily fluid intake of 1,700Ϫ2,200 ml (25Ϫ30 ml/kg BW) in addition to some 300 ml oxidation water is balanced by a fluid discharge of approximately 1,500 ml as urine, about 100 ml in stools, roughly 600 ml as perspiration and some 400 ml as expired air. (s. fig. 16.1) About 60% of the body's weight (ca. 55% in women) consists of water. The reservoir of body water is distributed between the intracellular space (ca. 40% of BW) and the extracellular space (ca. 20% of BW). The extracellular compartment consists of plasma fluids (ca. 4% of BW) and interstitial water (ca. 16% of BW), the latter also containing transcellular water (ca. 2.5% of BW). Because of its high degree of permeability, the body water is evenly shared between the intracellular and extracellular compartments. The water distribution between plasma and interstitium, regulated by Starling's forces, depends on the hydrostatic and colloidosmotic pressure gradients along the capillary walls. • Disturbances in the excretion of water are derived from (1.) an increase in ADH activity, (2 ( ( .) a reduction in distal filtrates available in the nephron, and (3.) greater absorption of wat...

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“…Επιπρoσθέτως, έχουν βρεθεί αυξηµένα τα επίπεδα του παράγοντα νέκρωσης όγκου (TNF-a), της ιντερλευκίνης-1β (IL-1β), της ιντερλευκίνης-2 (IL-2), του υποδοχέα της ιντερλευκίνης-2 παρούσα µελέτη αποτελείται από δύο επιµέρους σκέλη. Η πρώτη µελέτη βασίστηκε στην δυσκολία που υπάρχει στην καθηµερινή κλινική πρακτική για την ταυτοποίηση της υπεζωκοτικής συλλογής[88][89][90][91][92][93][94][95][96][97] .Ηρακλείου. Η µέση ηλικία των ασθενών που πληρούσαν κριτήρια εισαγωγής στη µελέτη ήταν 65 έτη (εύρος 32-92 έτη),71 άνδρες και 29 δύο ασθενείς ήταν χρόνια νεφρική ανεπάρκεια και σε δύο ασθενείς η κίρρωση του ήπατος.…”

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Μελέτη Των Κυτταροκινών Του Πλευριτικού Υγρού

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Ascitic fluid ?1-antitrypsin

Cited by 15 publications

References 19 publications

Biochemical analysis of ascitic (peritoneal) fluid: what should we measure?

Biochemical analysis of ascitic (peritoneal) fluid: what should we measure?

Oedema and ascites

Μελέτη Των Κυτταροκινών Του Πλευριτικού Υγρού

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