Assessment of the 12-Lead Electrocardiogram as a Screening Test for Detection of Cardiovascular Disease in Healthy General Populations of Young People (12–25 Years of Age)

Maron, Barry J.; Friedman, Richard A.; Kligfield, Paul; Levine, Benjamin D.; Viskin, Sami; Chaitman, Bernard R.; Okin, Peter M.; Saul, J. Philip; Salberg, Lisa; Hare, George F. Van; Soliman, Elsayed Z.; Chen, Jersey; Matherne, G. Paul; Bolling, Steven F.; Mitten, Matthew J.; Caplan, Arthur L.; Balady, Gary J.; Thompson, Paul D.

doi:10.1016/j.jacc.2014.05.006

Cited by 191 publications

(161 citation statements)

References 255 publications

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“…An additional challenge, among 10 to 15 million athletes of various ages, is the fact that the incidence of sudden cardiac death is very low [20-22, 31, 34-36]. As a consequence of the low incidence of disease in the athletic population the current literature suggests that all cardiac imaging modalities are inadequate to justify their use as primary screening modalities in athletes, as well as the general population [2,37]. Outside the athletic community, the medical community has come to the conclusion that routine exams in asymptomatic persons does little to improve health outcomes, and is largely a misappropriation of time and money [38].…”

Section: Current Preparticipation Screeningmentioning

confidence: 99%

“…Outside the athletic community, the medical community has come to the conclusion that routine exams in asymptomatic persons does little to improve health outcomes, and is largely a misappropriation of time and money [38]. A screening electrocardiogram (ECG) or stress testing are currently not recognized as acceptable screening tools in asymptomatic people [2,21,39,40]. Even though screening tests in asymptomatic patients have no firm evidence of utility, these tests are commonly touted as acceptable screening tools for asymptomatic athletes primarily because of lower cost and access [21].…”

Section: Current Preparticipation Screeningmentioning

confidence: 99%

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Part I - Sudden Cardiac Death in Athletes Pathophysiologic Screening: A Transformative Solution to a Social Imperative

Paterick¹

2015

JESO

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Section: Current Preparticipation Screeningmentioning

confidence: 99%

Section: Current Preparticipation Screeningmentioning

confidence: 99%

Part I - Sudden Cardiac Death in Athletes Pathophysiologic Screening: A Transformative Solution to a Social Imperative

Paterick¹

2015

JESO

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“…Пациенты, испытывающие регулярные психоэмоциональные стрессовые воздействия, предъявляют жалобы на повышенную утомляемость, кардиалгии ноющего характера, длящиеся часами, ощущение перебоев в работе сердца, приступов сердцебиения, потерю сознания [7,24,25]. Однако необходимо учитывать тот факт, что в большинстве случаев пациенты, испытывающие хронические психоэмоциональные перегрузки, являются лицами работоспособного возраста и не заинтересованы в потере работы.…”

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“…При выполнении ЭКГ характерно выявление нарушений процессов реполяризации, нарушений проведения и ритма сердца на ЭКГ в покое [7,8,21,24]. При велоэргометрическом ЭКГ исследовании с достижением субмаксимальной нагрузки наиболее часто регистрируются (более 750 в сутки) наджелудочковая и желудочковая экстрасистолия (30%), а так же нарушения процессов реполяризации неишемического характера (40%).…”

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Cardiomyopathy Due to Chronic Psychoemotional Stress: Independent Disease or a Manifestation of Other Pathology of the Heart?

Bondarev¹,

Vasilenko²,

Efimov³

2015

СПНО (MPSE)

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Выполнен обзор отечественной и зарубежной литературы по проблеме не коронарогенных, невоспалительных изменений сердца под воздействием хронического психоэмоционального стресса. Описаны патофизиологические, биохимические, генетические, морфологические клинические и инструментальные особенности сердца, развивающиеся на фоне хронического стресса. Описаны возможности радионуклидной диагностики трофических и функциональных нарушений миокарда на клеточном и молекулярном уровнях под воздействием стресса. В связи с наличием всех признаков самостоятельной нозологической единицы, от наличия причины до морфологических и функциональных нарушений, изменения в сердце под воздействием хронического психоэмоционального стресса могут быть выделены в самостоятельное заболевание -стрессорную кардиомиопатию. Сделан вывод, что согласно современной классификации Американской кардиологической ассоциации (2006 г.) изменения в миокарде под воздействием хронического психоэмоционального стресса могут быть отнесены к первичной приобретенной кардиомиопатии. Saint-Petersburg, Russia (195271, 27, pr. Mechnikov, St. Petersburg, Russia), e-mail: railway-hospital@spb.ru A review of domestic and foreign literature on the problem not coronaral, not inflammatory changes of the heart under the influence of chronic psycho-emotional stress. Described pathophysiological, biochemical, genetic, morphological, clinical and instrumental features of the heart, developing on the background of chronic stress were discribed. The features of radionuclide diagnostics trophic and functional disorders of the myocardium at the cellular and molecular levels under stress were investigator. Due to the presence of all signs of independent nosological unit, cause to morphological and functional abnormalities, changes in heart under the influence of chronic psycho-emotional stress can be an independent disease -stress cardiomyopathy. It is concluded that according to the modern classification of the American heart Association (2006). Changes in the myocardium under the influence of chronic psycho-emotional stress can be classified as primary acquired cardiomyopathy. Keywords: chronic, psycho-emotional stress, heart, primary acquired cardiomyopathy, diagnostics Стрессорные воздействия на организм способны вызвать как хорошо изученные заболевания, такие как ИБС, ГБ, так и менее изученные, такие как кардиомиопатия. ИБС, ГБ являются не столько следствием непосредственного воздействия на миокард, как результатом реакции сосудистой системы, обмена липопротеидов на миокард, то есть вторичными изменениями. В отношении кардиомиопатий нет на сегодняшний день единого мнения. Согласно положению Международной классификации болезней 10 пересмотра

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Congenital Heart Disease

Pieles

Stuart

2016

IOC Manual of Sports Cardiology

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Objective: To assess the influence of the pulmonary annulus diameter after reconstruction of the right ventricular (RV) outflow tract at repair of tetralogy of Fallot on pulmonary regurgitation and RV pressure load; and to evaluate the impact of pulmonary regurgitation on RV size and function. Setting: Paediatric cardiology and diagnostic radiology departments of a tertiary referral centre. Patients: 67 patients were examined at a median of 4.8 years after repair of tetralogy of Fallot by means of biplane angiocardiography and magnetic resonance imaging (MRI). Main outcome measures: Pulmonary annulus diameter and area, pulmonary regurgitant fraction, RV to left ventricular (LV) systolic pressure ratio, RV end diastolic volume, and RV ejection fraction were assessed.Results: There was a significant positive correlation between pulmonary annulus area indexed to body surface area and pulmonary regurgitation (angiocardiography: r = 0.55, p < 0.001; MRI: r = 0.59, p < 0.001). No significant correlation was found between pulmonary annulus index and RV to LV systolic pressure ratio even in patients with small pulmonary annulus areas (r = −0.24, NS). Pulmonary regurgitant fraction was positively correlated with normalised RV end diastolic volume (angiocardiography: r = 0.42, p < 0.05; MRI: r = 0.56, p < 0.01). RV ejection fraction decreased with increasing pulmonary regurgitation (angiocardiography: r = −0.42, p < 0.05; MRI: r = −0.41, p < 0.05). Conclusions: The extent of pulmonary regurgitation after tetralogy of Fallot repair correlates with the postoperative size of the pulmonary annulus and is closely correlated with the enlargement of the RV. An enlargement of the pulmonary annulus to the second lower standard deviation of normal results in a decrease of pulmonary regurgitation and is sufficient to achieve adequate RV pressure unloading.

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Assessment of the 12-Lead Electrocardiogram as a Screening Test for Detection of Cardiovascular Disease in Healthy General Populations of Young People (12–25 Years of Age)

Cited by 191 publications

References 255 publications

Part I - Sudden Cardiac Death in Athletes Pathophysiologic Screening: A Transformative Solution to a Social Imperative

Part I - Sudden Cardiac Death in Athletes Pathophysiologic Screening: A Transformative Solution to a Social Imperative

Cardiomyopathy Due to Chronic Psychoemotional Stress: Independent Disease or a Manifestation of Other Pathology of the Heart?

Congenital Heart Disease

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