Association of Isolated Polyhydramnios at or Beyond 34 Weeks of Gestation and Pregnancy Outcome

Abstract

Isolated polyhydramnios at admission for labor at or beyond 34 weeks of gestation is associated with adverse obstetric and neonatal outcomes.

Amount of polyhydramnios attributable to diabetes may be less than previously reported

“…Up to 2% of all pregnancies have an excess amount of fluid meeting the criteria for polyhydramnios[1,2]. It is reported in the literature that approximately sixty percent of polyhydramnios is idiopathic; twenty percent of polyhydramnios can be attributed to poorly controlled or undiagnosed diabetes and the remainder of cases of polyhydramnios are associated with fetal anomalies[2-4]. Several adverse outcomes have been associated with polyhydramnios including preterm labor and rupture of membranes.…”

Can the Myocardial Performance Index Be Used as a New Predictive Factor for a Poor Prognosis in Fetuses With Idiopathic Polyhydramnios?

“…Polyhydramnios is defined as excessive accumulation of amniotic fluid and may be associated with fetal infections and anomalies of the gastrointestinal, cardiac, and central nervous systems, placental tumors, multiple gestations, and maternal diabetes. Irrespective of etiology, the overall incidence ranges from 0.2% to 2% 1 . Idiopathic polyhydramnios is a diagnosis of exclusion, and it accounts for approximately 50% to 60% of cases 1 …”

“…Irrespective of etiology, the overall incidence ranges from 0.2% to 2% 1 . Idiopathic polyhydramnios is a diagnosis of exclusion, and it accounts for approximately 50% to 60% of cases 1 …”

“…Idiopathic polyhydramnios was found to be an independent risk factor for adverse perinatal outcomes, such as fetal distress, shoulder dystocia, cesarean delivery, and respiratory distress syndrome (RDS) 1 . The possible proposed mechanism was alterations in fetal oxygen metabolism and circulation in polyhydramniotic fetuses 2 .…”

“…shoulder dystocia, cesarean delivery, and respiratory distress syndrome (RDS). 1 The possible proposed mechanism was alterations in fetal oxygen metabolism and circulation in polyhydramniotic fetuses. 2 However, because of a lack of definitive studies, the role of antenatal surveillance in pregnancies complicated by idiopathic polyhydramnios is uncertain.…”