Associations between Adverse Childhood Experiences and Emergency Department Utilization in an Adult Medicaid Population

Scott, Kristin L.; Cohen-Cline, Hannah

doi:10.3390/ijerph191610149

Cited by 3 publications

(1 citation statement)

References 53 publications

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“…Some studies have previously examined ACEs and SES separately or examined SES as a mediating factor, linking inaccessibility of resources to negative health consequences. However, more recent studies, especially in light of the COVID-19 pandemic, have considered ACEs inclusive of SES, rather than examining them as separate factors; this is due to how low SES is so often co-morbid with high ACEs and can be considered a negative household experience, a category of ACEs [ 10 , 11 ]. For instance, in literature examining the currently defined ACEs scale, low socioeconomic status is classified as an impactful form of trauma consistent among various racial and ethnic groups, classifying it as an ACE [ 12 ].…”

Section: Introductionmentioning

confidence: 99%

Adverse Childhood Experiences (ACEs) and Environmental Exposures on Neurocognitive Outcomes in Children: Empirical Evidence, Potential Mechanisms, and Implications

Gladieux

Gimness

Rodriguez

et al. 2023

Toxics

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The purpose of this article is to examine the current literature regarding the relationship between adverse childhood experiences (ACEs) and environmental exposures. Specifically, the paper will focus on how this relationship between ACEs and physical environmental factors impacts the neurocognitive development of children. With a comprehensive literary search focusing on ACEs, inclusive of socioeconomic status (SES), and environmental toxins common in urban environments, the paper explores how these factors contribute to cognitive outcomes that are associated with the environment and childhood nurturing. The relationship between ACEs and environmental exposures reveals adverse outcomes in children’s neurocognitive development. These cognitive outcomes include learning disabilities, lowered IQ, memory and attention problems, and overall poor educational outcomes. Additionally, potential mechanisms of environmental exposures and children’s neurocognitive outcomes are explored, referencing data from animal studies and evidence from brain imaging studies. This study further analyzes the current gaps in the literature, such as the lack of data focusing on exposure to environmental toxicants resulting from experiencing ACEs and discusses the research and social policy implications of ACEs and environmental exposure in the neurocognitive development of children.

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Section: Introductionmentioning

confidence: 99%

Adverse Childhood Experiences (ACEs) and Environmental Exposures on Neurocognitive Outcomes in Children: Empirical Evidence, Potential Mechanisms, and Implications

Gladieux

Gimness

Rodriguez

et al. 2023

Toxics

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Impact of socioeconomic factors on allergic diseases

Perry,

Grant,

Dantzer

et al. 2024

Journal of Allergy and Clinical Immunology

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Early life adversity increases risk for chronic posttraumatic pain, data from humans and rodents

McKibben,

Woolard,

McLean

et al. 2024

Preprint

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Traumatic stress exposures (TSE) are common in life. While most individuals recover following a TSE, a substantial subset develop adverse posttraumatic neuropsychiatric sequelae such as chronic posttraumatic musculoskeletal pain (CPMP). Vulnerability factors for CPMP are poorly understood, which hinders identification of high-risk individuals for targeted interventions. One known vulnerability factor for many pain types is exposure to early life adversity (ELA), but few studies have assessed whether ELA increases risk for CPMP. This study used data from the AURORA study, a prospective human cohort study of TSE survivors, to test the hypothesis that ELA increases risk for CPMP. In addition, in secondary analyses, we assessed which subtypes of ELA (including childhood bullying) were most predictive of CPMP and whether a rat ELA model consisting of neonatal limited bedding (NLB), combined with single prolonged stress (SPS) in adulthood, would accurately model human findings. In AURORA study participants (n=2,480), using multinomial logistic regression modeling of four identified latent pain classes, we found that ELA increased vulnerability to the high unremitting pain class (OR=1.047, p<0.001), the moderate pain class (OR=1.031, p<0.001), and the moderate recovery pain class (OR=1.018, p=0.004), with physical abuse, emotional abuse, and bullying being the strongest predictors of high pain class assignment. Similarly, in male and female Sprague Dawley rats, in comparison to SPS alone NLB combined with SPS caused increased baseline sensitivity and prolonged mechanical hypersensitivity (F(11,197)=3.22, p<0.001). Further studies in animals and humans are needed to understand mechanisms by which ELA confers vulnerability to CPMP.

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Associations between Adverse Childhood Experiences and Emergency Department Utilization in an Adult Medicaid Population

Cited by 3 publications

References 53 publications

Adverse Childhood Experiences (ACEs) and Environmental Exposures on Neurocognitive Outcomes in Children: Empirical Evidence, Potential Mechanisms, and Implications

Adverse Childhood Experiences (ACEs) and Environmental Exposures on Neurocognitive Outcomes in Children: Empirical Evidence, Potential Mechanisms, and Implications

Impact of socioeconomic factors on allergic diseases

Early life adversity increases risk for chronic posttraumatic pain, data from humans and rodents

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