Athletic Heart Remodeling in Athletes with Arterial Hypertension

Features of cardiac remodeling depending on the mode of training session

“…This is due to increased cardiac output during exercise, i.e. LV volume overload as well as increased systemic blood pressure, and eccentric LV hypertrophy develops without change in the ratio of LV wall thickness to its diameter [7]. In this case, hypertrophy is the result of addition of sarcomeres in series and longitudinal cell growth [17].…”

“…The impact of volume remains relatively stable, but it may decrease during high-intensity training as a result of rising after exercise [2]. Athletes, whose trainings are dominated by static or isometric loads (weightlifters, wrestlers, throwers), develop concentric hypertrophy with increasing LV wall thickness, without changes in the size of its cavity, which is caused by an increased heart afterload resulted from increased systemic blood pressure during exercises [7]. Although the prevalence of high blood pressure is approximately 50 % lower in athletes than in the general population, it is also the most common cardiovascular condition in athletes [25].…”

“…Although the prevalence of high blood pressure is approximately 50 % lower in athletes than in the general population, it is also the most common cardiovascular condition in athletes [25]. Possibly, myocardial hypertrophy in athletes may be caused by increased blood pressure, which is confirmed by a number of studies that have shown a large majority of athletes with hypertensive stress response LV myocardial mass than athletes with normotensive reaction [7].…”

“…The experience in athletes with concentric remodeling was 8.5 years (7.0-10.0 years), and with eccentric hypertrophy -6.0 years (3.0-18.0 years), no statistically significant differences were obtained. The authors hypothesized, that the development of the most prognostically unfavorable variant -concentric LV hypertrophy -directly depends on the time of exposure to strength loads [6].…”