Atrial Components Contributing to Pseudo r' Deflection in Lead V1 in Slow/Fast Atrioventricular Nodal Reentrant Tachycardia. Analysis of the Atrial Activation Sequence by Basket Catheter Isochronal Mapping.

Kuga, Keisuke; Li, Aiyan; Endo, M; Niho, Bumpei; Suzuki, Akihiro; Enomoto, Mami; Kanemoto, Miyako; Yamaguchi, Iwao

doi:10.1253/circj.66.236

Cited by 5 publications

(6 citation statements)

References 8 publications

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“…Twelve-lead ECGs were analyzed for the rate of clinical AVNRT, presence of pseudo-r 0 deflection in lead V 1 , pseudo-S wave in inferior leads, P-in-QRS pattern (absence of pseudo-r 0 deflection in lead V 1 and/or pseudo-S wave in inferior leads), and QRS alternans. 11 Five patients were excluded for the analysis: 3 with slow/slow AVNRT, 1 with fast/slow AVNRT, and 1 with left variant slow/fast AVNRT.…”

Section: Definition Of Ecg Parametersmentioning

confidence: 99%

High prevalence of concealed Brugada syndrome in patients with atrioventricular nodal reentrant tachycardia

Hasdemir¹,

Payzın²,

Kocabaş³

et al. 2015

Heart Rhythm

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Section: Definition Of Ecg Parametersmentioning

confidence: 99%

High prevalence of concealed Brugada syndrome in patients with atrioventricular nodal reentrant tachycardia

Hasdemir¹,

Payzın²,

Kocabaş³

et al. 2015

Heart Rhythm

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“…3 Analysis of the atrial activation sequence during AVNRT in humans revealed that the pseudo-r' deflection in lead V 1 is determined by activation of the superolateral aspect of the right atrium. 26 As a result of this finding, we hypothesize that higher prevalence of pseudo-r' deflection in patients DI-Type 1…”

Section: Electrocardiographic Variables Associated With Underlying Br...mentioning

confidence: 81%

Electrocardiographic variables associated with underlying Brugada syndrome or drug‐induced Type 1 Brugada pattern in patients with slow/fast atrioventricular nodal reentrant tachycardia

Hasdemir

Şahin

Duran

et al. 2022

Journal of Arrhythmia

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Background The coexistence of clinical atrioventricular nodal reentrant tachycardia (AVNRT) and drug‐induced type 1 Brugada pattern (DI‐Type 1 BrP) has been previously reported. The present study was designed to determine the 12‐lead ECG characteristics at baseline and during AVNRT and to identify a subset of 12‐lead ECG variables of benefit associated with underlying Brugada syndrome (BrS)/DI‐Type 1 BrP among patients with slow/fast AVNRT. Methods A total of 40 (11 numerical/29 categorical) 12‐lead ECG parameters were analyzed and compared between patients with (n = 69) and without (n = 104) BrS/DI‐Type1‐BrP matched for age, female gender, body mass index, left ventricular ejection fraction and comorbid conditions. Five distinct types of ECG pattern (Type A/B/C/D/E) in V1–V2 leads during AVNRT were defined. Results A total of nine electrocardiographic variables, four at baseline, and five during AVNRT were identified. At baseline, patients with BrS/DI‐Type 1 BrP had higher prevalence of interatrial block, leftward shift of frontal plane QRS axis, the absence of normal QRS pattern (the presence of rSr’ pattern or type 2/3 Brugada pattern) in V1–V2 and QRS fragmentation in inferior leads compared to patients without BrS/DI‐Type 1 BrP. During AVNRT, patients with BrS/DI‐Type 1 BrP had higher prevalence of Type A ECG pattern (“coved‐type” ST‐segment elevation) in V1–V2, Type C ECG pattern (pseudo‐r’ deflection in V1 and “RBBB‐like” pattern in V2), pseudo‐r’ deflection in V1, QRS fragmentation in inferior leads and “isolated” QRS fragmentation/notching/slurring in aVL compared to patients without BrS/DI‐Type 1 BrP. Conclusions We identify several electrocardiographic variables that point to an underlying type 1 BrP among patients with slow/fast AVNRT.

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“…This demonstrates a low to high activation pattern suggesting that the origin of the P wave is near the atrioventricular (AV) node. The P wave is inscribed on the end of the QRS complex demonstrating a short ventriculoatrial (VA) time, making atrioventricular reentrant tachycardia (AVRT) unlikely and would be the hallmark of atrioventricular nodal reentrant tachycardia (AVNRT) in the context of a regular narrow complex tachycardia 1 . The organized atrial activity demonstrated by the retrograde P waves demonstrates that the rhythm is not AF; it can also be seen that the variability in cycle length is due to the variability in the AV time with a fixed VA time, which rules out atrial tachycardia.…”

Section: Discussionmentioning

confidence: 99%

Irregular Narrow Complex Tachycardia: What Is the Mechanism?

Temple¹,

Fitchet

Fox³

2012

Pacing Clinical Electrophis

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Atrial Components Contributing to Pseudo r' Deflection in Lead V1 in Slow/Fast Atrioventricular Nodal Reentrant Tachycardia. Analysis of the Atrial Activation Sequence by Basket Catheter Isochronal Mapping.

Cited by 5 publications

References 8 publications

High prevalence of concealed Brugada syndrome in patients with atrioventricular nodal reentrant tachycardia

High prevalence of concealed Brugada syndrome in patients with atrioventricular nodal reentrant tachycardia

Electrocardiographic variables associated with underlying Brugada syndrome or drug‐induced Type 1 Brugada pattern in patients with slow/fast atrioventricular nodal reentrant tachycardia

Irregular Narrow Complex Tachycardia: What Is the Mechanism?

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