Atrial endocardial activation in man electrode catheter technique for endocardial mapping

Josephson, Mark E.; Scharf, Daniel L.; Kastor, John A.; Kitchen, James G.

doi:10.1016/s0002-9149(77)80210-5

Cited by 80 publications

(20 citation statements)

References 16 publications

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“…Previous studies based on M-mode echocardiography and invasive catheterization reported an inter-atrial delay of 25 ms and 20 ms, respectively, in normal subjects [28,29]. Since these studies were performed in a small number of normal controls (20 or less) [27,29], this study adds further confirmatory information over a large control population.…”

Section: Interatrial Delay In Atrial Contractionsupporting

confidence: 59%

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Tissue Doppler echocardiographic evidence of atrial mechanical dysfunction in coronary artery disease

Fung

Zhang

et al. 2005

International Journal of Cardiology

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Section: Interatrial Delay In Atrial Contractionsupporting

confidence: 59%

“…The interatrial delay was 24 ms. This represents the propagation of the atrial depolarization from the sinoatrial node to the LA [27]. Previous studies based on M-mode echocardiography and invasive catheterization reported an inter-atrial delay of 25 ms and 20 ms, respectively, in normal subjects [28,29].…”

Section: Interatrial Delay In Atrial Contractionmentioning

confidence: 91%

Tissue Doppler echocardiographic evidence of atrial mechanical dysfunction in coronary artery disease

Fung

Zhang

et al. 2005

International Journal of Cardiology

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“…Several invasive electrophysiologic techniques have been used to measure interatrial electrical conduction time [15, 16]. Rokas et al [17]showed an increase in interatrial electrical conduction time in patients with PSS (70 ms in PSS patients vs. 37 ms in controls), which was measured as conduction time from the RA to the portion of the coronary sinus behind the left atrium using catheters.…”

Section: Discussionmentioning

confidence: 99%

Atrial Involvement in Patients with Progressive Systemic Sclerosis: Relationship between Ultrasonic Tissue Characterization of the Atrium and Interatrial Conduction

Mizuno¹,

Fujimoto

Nakano

et al. 1999

Cardiology

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Objective: The aim of this study was to assess atrial lesions using ultrasonic tissue characterization and to determine the contribution of atrial lesions to the interatrial electromechanical coupling conduction time in patients with progressive systemic sclerosis (PSS). Methods: Twenty patients with PSS and 20 age-matched healthy controls were evaluated. The cyclic variation in integrated backscatter value (CV-IB) was measured at the interatrial septum (IAS) from apical four chamber view. M-modes of ventricular long axis motion along with phono- and electrocardiograms were recorded simultaneously at the right lateral (RT) and left lateral (LT) sites of the atrioventricular (AV) rings and central fibrous body (CFB) in the apical four-chamber view. Intervals from the P wave on ECG to the echocardiographic onset of atrial contraction as a point of inflection in long axis M-mode echocardiogram were measured at the RT and LT sites of AV rings and CFB (P-RT, P-LT, P-SEP, respectively). Interatrial electromechanical coupling conduction time was determined as [(P-LT) – (P-RT)]. Results: In patients with PSS compared to normal controls, P-RT, P-SEP, P-LT, and interatrial conduction time were greater, while CV-IB in IAS decreased. Furthermore, CV-IB in IAS correlated well with interatrial conduction time (r = 0.7, p < 0.01) in patients with PSS. Conclusions: Interatrial electromechanical coupling times may be prolonged due to atrial tissue damage in patients with PSS.

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“…50) It is considered that the patients with atrial fibrillation show abnormal dispersion of the atrial refractory period. 51) Josephson et al 52) reported that the difference in the effective refractory period between the high right atrium and the ostium of coronary sinus is 35 ms in patients with atrial fibrillation and 5 ms in those without atrial fibrillation. A dispersion of refractory period is already present in the atrium of the patients with atrial fibrillation.…”

Section: ) Effect Of Atrial Pacing Sitementioning

confidence: 99%

Pacing Therapy for Prevention of Atrial Fibrillation

Mizutani

2005

J Arrhythmia

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Nonpharmacological therapy is being evaluated for the prevention of atrial fibrillation (AF). Pacing has been proposed as a one of the option to prevent AF. In patients with bradycardia and requiring pacemaker, physiological pacing such as dual chamber pacing or atrial pacing has an advantage against ventricular pacing in prevention AF. Pacing from novel site like the dual-site atrium, biatrial, Bachman, and low sepatl pacing may reduce AF burden and new anti-AF pacemakers with atrial prevention algorithm may decrease AF further. However, selecting the appropriate patients and adopting tailored-therapy for individual patient is likely to remain one of the difficulties in achieving an advantage. This review discusses the current status of pacing therapy for the prevention of AF. It also discusses the some of merits and limitations of pacing therapy for the treatment of AF. (J Arrhythmia 2005; 21: 438-449)

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Atrial endocardial activation in man electrode catheter technique for endocardial mapping

Cited by 80 publications

References 16 publications

Tissue Doppler echocardiographic evidence of atrial mechanical dysfunction in coronary artery disease

Tissue Doppler echocardiographic evidence of atrial mechanical dysfunction in coronary artery disease

Atrial Involvement in Patients with Progressive Systemic Sclerosis: Relationship between Ultrasonic Tissue Characterization of the Atrium and Interatrial Conduction

Pacing Therapy for Prevention of Atrial Fibrillation

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