Atrial fibrillation in endurance-trained athletes: Table 1

Sorokin, Alexey V.; Araújo, Claudio Gil; Zweibel, Steven; Thompson, Paul D.

doi:10.1136/bjsm.2009.057885

Cited by 73 publications

(39 citation statements)

References 37 publications

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“…However, the debate is of great relevance to the individual athlete who has profound myocardial remodeling or arrhythmias and needs us to understand the mechanism so that we can target our treatments and best prevent adverse outcomes. At present there is insufficient evidence to treat athletes with arrhythmias any differently from the general population, 80 but if we start to accept that exercise may contribute to the disease substrate then it would seem relevant to assess whether changes in exercise practice may change clinical outcomes. We need open-minded investigators with the patience, will, and resources to conduct adequately powered prospective studies.…”

Section: Resultsmentioning

confidence: 99%

Can Intensive Exercise Harm the Heart?

2014

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Section: Resultsmentioning

confidence: 99%

Can Intensive Exercise Harm the Heart?

2014

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“…Likely reasons include changes in autonomic drive including increased parasympathetic modulation at rest and increased sympathetic modulation during exercise, increased atrial size and increased inflammation (Sorokin et al 2009). …”

Section: Potential Arrhythmic Substrate(s) For Afmentioning

confidence: 99%

Cardiovascular function and the veteran athlete

et al. 2010

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The cardiovascular benefits of exercise are well known. In contrast, the impact of lifelong endurance exercise is less well understood. Long-term high-intensity endurance exercise is associated with changes in cardiac morphology together with electrocardiographic alterations that are believed to be physiologic in nature. Recent data however has suggested a number of deleterious adaptive changes in cardiac structure, function and electrical activity, together with peripheral and cerebral vascular structure and function. This review serves to detail knowledge in relation to; (1) Cardiac structure and function in veteran endurance athletes focusing on the differentiation of physiological and pathological changes in cardiac remodelling; (2) Cardiac electrical activity and the veteran endurance athlete with attention to arrhythmias, the substrate for arrhythmia generation and the clinical significance of such arrhythmias; (3) Peripheral and cerebral vascular structure and function in ageing and endurance-trained individuals; and (4) directions for future research.

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“…100 -103 This appears to be particularly true of the older athletic patient. The mechanisms producing atrial fibrillation in athletes are speculative, but a combination of exercise-induced left atrial remodeling and inflammation, 69,104 increased sympathetic activity during exercise, 100 and parasympathetically mediated slow resting heart rates potentiating atrial escape 93 has been suggested as a potential contributor.…”

Section: Arrhythmiamentioning

confidence: 99%