2008
DOI: 10.1111/j.1540-8159.2009.02180.x
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Atrial Flutter with Spontaneous 1:1 Atrioventricular Conduction in Adults: An Uncommon but Frequently Missed Cause for Syncope/Presyncope

Abstract: Aims:To compare patients with atrial flutter (AFl) 220/min. (PACE 2009; 32:82-90) atrial flutter, AV conduction, ICD; syncope, tachycardia

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Cited by 22 publications
(12 citation statements)
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“…In addition, 8.2% of episodes of inappropriate shocks had cycle lengths of <250 ms. In previous studies, the mean ventricular cycle length during atrial flutter with 1:1 atrioventricular (AV) conduction was between 265 ms and 292 ms. 26) 27) Enhanced AV nodal conduction in this young population, together with a short, heightened sympathetic tone that leads to shortening of the atrial flutter cycle length might have contributed to the very short cycle length of the SVAs. In our patient population, two patients with BS had three VT episodes with a cycle length of 282.0±11.7 ms, which activated ICD shocks.…”
Section: Discussionmentioning
confidence: 97%
“…In addition, 8.2% of episodes of inappropriate shocks had cycle lengths of <250 ms. In previous studies, the mean ventricular cycle length during atrial flutter with 1:1 atrioventricular (AV) conduction was between 265 ms and 292 ms. 26) 27) Enhanced AV nodal conduction in this young population, together with a short, heightened sympathetic tone that leads to shortening of the atrial flutter cycle length might have contributed to the very short cycle length of the SVAs. In our patient population, two patients with BS had three VT episodes with a cycle length of 282.0±11.7 ms, which activated ICD shocks.…”
Section: Discussionmentioning
confidence: 97%
“…[1] However, accessory pathways can have shorter refractory periods than the AV node, thereby circumventing the AV node's protective delaying properties and allowing for more rapid conduction to the ventricles. [1][2][3] While normal conduction through the AV node and His-Purkinje system results in synchronized ventricular depolarization and thus a narrow QRS complex, most accessory pathways insert directly into the ventricular myocardium and conduct in an aberrant fashion, causing the ventricles to contract out of sync and thereby producing a wide QRS complex. In addition to direct conduction from the atria to the ventricles, accessory pathways can also cause wide complex tachycardias by forming reentrant circuits.…”
Section: Discussionmentioning
confidence: 99%
“…AFL was reported as an early sign of the cardiac complications of type 1 MD in childhood . In patients without type 1 MD, the association of 1/1 AFL with sympathetic hyperactivity, such as exercise, was well documented, either in untreated patients or in patients receiving antiarrhythmic drugs. Exercise allowed all the atrial impulses to pass through the AV node to the ventricles with a 1/1 conduction.…”
Section: Discussionmentioning
confidence: 99%
“…Exercise allowed all the atrial impulses to pass through the AV node to the ventricles with a 1/1 conduction. Turitto et al recommended to be aware of the different presentations of 1/1 AFL to avoid misdiagnosis/mismanagement and to consider the diagnosis in patients with narrow or wide QRS tachycardia and rates above 220 beats/min.…”
Section: Discussionmentioning
confidence: 99%