2020
DOI: 10.15420/aer.2020.07
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Atrial Tachycardias After Atrial Fibrillation Ablation: How to Manage?

Abstract: With catheter ablation becoming effective for non-pharmacological management of AF, many cases of atrial tachycardia (AT) after AF ablation have been reported in the past decade. These arrhythmias are often symptomatic and respond poorly to medical therapy. Post-AF-ablation ATs can be classified into the following three categories: focal, macroreentrant and microreentrant ATs. Mapping these ATs is challenging because of atrial remodelling and its complex mechanisms, such as double ATs and multiple-loop ATs. Hi… Show more

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Cited by 16 publications
(8 citation statements)
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“…Our study also showed that the HPSD method could not reduce the risk of recurrent AFL or AT. This result could be caused by several factors: (i) different arrhythmogenic mechanisms, (ii) some patients received not only PVI, and (iii) AFL or AT could be caused by scar formation due to RFCA 51,53 …”
Section: Discussionmentioning
confidence: 99%
“…Our study also showed that the HPSD method could not reduce the risk of recurrent AFL or AT. This result could be caused by several factors: (i) different arrhythmogenic mechanisms, (ii) some patients received not only PVI, and (iii) AFL or AT could be caused by scar formation due to RFCA 51,53 …”
Section: Discussionmentioning
confidence: 99%
“…Not only recurrent AF requires redo ablation, but atypical atrial flutter and atrial tachycardia after AF ablation also lead to redoing ablation in up to 8% of cases. 52,53 The low recurrence rate of those AAs beyond the blanking period reflects the higher success rate of AF ablation. FPI is another outcome that represented the efficacy of AF ablation.…”
Section: Major Complicationsmentioning
confidence: 99%
“…In addition, the patient's history of recent AF ablation was key in diagnosing the arrhythmia, as the incidence of AT post AF ablation is 5 Mobitz 40%. 2,3 There are various causes of aberrated conduction: (1) pre-existing and/or physiologic/functional bundle branch or interventricular conduction block, (2) metabolic derangements (i.e., hyperkalemia), and (3) druginduced (i.e., propafenone toxicity). 4 The described mechanisms of functional/physiologic block include acceleration-dependent block above the critical heart rate, phase 3 block of the action potential, phase 4 bradycardia-dependent block due to disease in the His-Purkinje system, and transseptal, retrograde, concealed invasion of the bundle branch (BB) rendering it refractory to subsequent depolarizations.…”
Section: Commentarymentioning
confidence: 99%