Author Correction: Excitotoxic inactivation of constitutive oxidative stress detoxification pathway in neurons can be rescued by PKD1

Abstract

The original version of this Article contained an error in the spelling of the author Álvaro Sebastián-Serrano, which was incorrectly given as Álvaro Sebastián Serrano. This has now been corrected in both the PDF and HTML versions of the Article.

Bioactive Nanoenzyme Reverses Oxidative Damage and Endoplasmic Reticulum Stress in Neurons under Ischemic Stroke

“…68 In addition, as a basic element necessary for the human body, manganese is an essential metal catalyst for certain metal enzymes, including SOD2. 69 Among the enzymes having manganese as the active metal center, SOD2 is particularly associated with the scavenging of free radicals. 70 According to our previous manganese ion release experiment, HSA-Mn 3 O 4 can effectively release manganese ion under a high ROS level.…”

“…It provides significant antioxidant enzymes in the central nervous system and is considered to be involved in protection mechanisms that cells employ against reactive oxygen species injury following ischemic stroke. , According to a previous study, knocking down SOD2 highly aggravates cerebral infarction after ischemia stroke . In addition, as a basic element necessary for the human body, manganese is an essential metal catalyst for certain metal enzymes, including SOD2 . Among the enzymes having manganese as the active metal center, SOD2 is particularly associated with the scavenging of free radicals .…”