Barrettʼs Esophagus and Achalasia. A Case Report

Lee, F I; Bellary, S.V.

doi:10.1097/00004836-199110000-00017

Cited by 6 publications

(2 citation statements)

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“…Further evidence that achalasia can develop in the setting of chronic GORD is provided by the observation that 3 of our patients who complained of chronic heartburn had Barrett's oesophagus on their initial endoscopic examination.13 Barrett's oesophagus has been well described as a consequence of the severe GORD that can accompany the surgical treatment of achalasia, but has been described rarely in untreated patients.14 15 Although we cannot exclude the possibility that oesophageal irritation from retained material caused the columnar metaplasia in these patients, it seems more likely that achalasia appeared after Barrett's oesophagus had developed in the typical setting of chronic GORD.…”

Section: Discussionmentioning

confidence: 83%

Heartburn in patients with achalasia.

Spechler

Souza

Rosenberg

et al. 1995

Gut

104

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Section: Discussionmentioning

confidence: 83%

Heartburn in patients with achalasia.

Spechler

Souza

Rosenberg

et al. 1995

Gut

104

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“…Previously published cases of the coexistent BE and achalasia in patients without prior surgical myotomy or pneumatic dilation is presented in Table 1 [7][8][9][10][11][12][13]. Based on English literature review, this appears to be the tenth case reported.…”

Section: Discussionmentioning

confidence: 99%

Barrett’s esophagus following treatment of achalasia with botulinum toxin

Wells¹,

Carpenter²,

Huguet³

et al. 2012

OJGas

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Achalasia is an uncommon primary motor disorder of the esophagus with an annual incidence of 0.8/100,000. Very few cases of coexistent Barrett's esophagus (BE) and achalasia in patients without prior surgical myotomy or pneumatic dilation have been reported. We report the case of a 65-year-old female who was diagnosed with achalasia in June 2002. Endoscopy at that time revealed biopsy-confirmed normal esophageal mucosa. The patient subsequently underwent two trials of botox injection with progressively worsening dysphagia. A repeat endoscopy two years later showed a short segment of salmon-colored mucosa in the distal esophagus which was biopsy-confirmed Barrett's epithelium with no dysplasia. The patient eventually underwent laparoscopic Heller myotomy and Toupet fundoplication. Postoperatively, she recovered well and with significant alleviation of her dysphagia. This study reviews reported cases of coexistent achalasia and BE, and discusses possible etiologies of concurrent BE and achalasia, and implications for treatment.

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Reproducibility of ambulatory esophageal pH monitoring in the aperistaltic esophagus

et al. 1996

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The reproducibility of ambulatory 24-h esophageal pH monitoring was evaluated in 16 consecutive patients by comparing the difference in two consecutive 24-h periods. The study group included 8 patients with scleroderma esophagus and 8 treated achalasia patients. The amount of reflux was expressed as the percentage of time the pH was < 4.0. Both groups demonstrated excellent intrapatient reproducibility overall: 96% in scleroderma patients and 95% in those patients with achalasia. The least concordance was found in the lengths of the longest reflux events-70% when supine in scleroderma patients and 59% when upright in patients with achalasia. There was no significant difference (p > 0.05) between day 1 and day 2 for either group of patients for any of the elements studied. These results indicate that intrapatient variability of gastroesophageal reflux in patients with scleroderma esophagus and treated patients with achalasia is very low and following therapeutic intervention, a high level of confidence can be placed in subsequent pH monitoring as an indicator of treatment effect.

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Barrettʼs Esophagus and Achalasia. A Case Report

Cited by 6 publications

References 0 publications

Heartburn in patients with achalasia.

Heartburn in patients with achalasia.

Barrett’s esophagus following treatment of achalasia with botulinum toxin

Reproducibility of ambulatory esophageal pH monitoring in the aperistaltic esophagus

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