Benign intracranial hypertension: A review of 79 cases in infancy and childhood

Grant, D. N.

doi:10.1136/adc.46.249.651

Cited by 108 publications

(47 citation statements)

References 12 publications

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“…Grant 13 , followin-up 79 children with IIH, showed that papilledema disappeared in 48% of the cases after 2 months, in 54% after 6 months, in 67% after one year, in 75% after two years, in 85% after 3 years and in 86% after 10 years. The optic atrophy was only observed respectively in two patients two and four years after the beginning of the treatment 13 .…”

Section: Discussionmentioning

confidence: 99%

“…The main abnormality disclosed by examination is papilledema, occasionally with hemorrhagic exsudates 13 . However, its absence does not exclude IIH 2,13,[18][19][20][21][22] .…”

Section: Discussionmentioning

confidence: 99%

“…Paralysis of the abducent nerve, trochlear and facial paralysis 23 are also common findings. Less frequently nystagmus, bilateral intranuclear ophthalmoplegia, dissociation of pupilary reflex 24 and limi- tation of the upper gaze may occur 13 . Rarely the patient may reveal ataxia 12,13 and the presence of a Babinski sign 12 .…”

Section: Discussionmentioning

confidence: 99%

“…The most commom symptoms are: headache, otalgia, diplopia, photopsia, vomiting, mialgia, dizzines and pulsatile tinnitus 12,13 . The typical characteristic of headache in the IIH are: severe intensity (93%); pulsating quality (83%); bilateral location (50%); associated to nausea and/or vomiting (57%), photophobia and phonophobia (30%) and photopsias (20%) with a daily frequency (73%) and lasting from four days to three years 2,[14][15][16] .…”

Section: Discussionmentioning

confidence: 99%

“…However, its absence does not exclude IIH 2,13,[18][19][20][21][22] . Paralysis of the abducent nerve, trochlear and facial paralysis 23 are also common findings.…”

Section: Discussionmentioning

confidence: 99%

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Long-term evolution of papilledema in idiopathic intracranial hypertension: observations concerning two cases

Piovesan

Lange

Piovesan

et al. 2002

Arq. Neuro-Psiquiatr.

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-Chronic headaches, associated with papilledema and pulsatile tinnitus without any neuroradiologic, cytobiochemical or cerebrospinal fluid abnormalities are suggestive of idiopathic intracranial hypertension (IIH). However the absence of the papilledema does not rule out this diagnosis. The reason why some patients do not develop papilledema in IIH is ignored, however there are some hypotheses concerning the structure of the optical nerve. In this study we described two female patients that presented diagnosis of IIH with papilledema, with subsequent resolution of papilledema without the due resolution of intracranial hypertension. The long-term behavior of the optic nerve (ON) facing an increased intracranial pressure was evaluated through repeated measurements of the intracranial pressure. We concluded that the ON submitted to high intracranial pressure for a certain lenght of time can adapt itself with subsequent disappearance of the papilledema. The presence or not of papilledema in IIH can be related to the period in which the diagnosis is accomplished.KEY WORDS: headache, intracranial pressure, papilledema, pseudotumor cerebri.Avaliação do comportamento do papiledema na hipertensão intracraniana idiopática: a propósito de Avaliação do comportamento do papiledema na hipertensão intracraniana idiopática: a propósito de Avaliação do comportamento do papiledema na hipertensão intracraniana idiopática: a propósito de Avaliação do comportamento do papiledema na hipertensão intracraniana idiopática: a propósito de Avaliação do comportamento do papiledema na hipertensão intracraniana idiopática: a propósito de dois casos dois casos dois casos dois casos dois casos RESUMO -Cefaléias com características crônica, diária, acompanhadas de edema de papila e tinitus pulsátil, sem nenhum achado neuroradiológico ou citobioquímico no líquor, são altamente sugestivas de hipertensão intracraniana idiopática (HII). Entretanto a ausência do papiledema não invalida o seu diagnóstico. A razão pela qual alguns pacientes não desenvolvem papiledema na HII é desconhecida, porém algumas hipóteses relacionadas com propriedades intrínsecas da bainha do nervo óptico têm sido propostas. Neste estudo relatamos dois pacientes do sexo feminino que apresentaram diagnóstico de HII com papiledema, evoluindo para resolução do papiledema sem a devida resolução da HII. O comportamento do nervo óptico (NO) frente ao aumento da pressão intracraniana foi avaliado neste estudo a partir de um monitoramento intermitente criterioso da pressão intracraniana. Concluímos que o NO submetido a um período de hipertensão intracraniana pode adaptar-se a este ambiente promovendo uma resolução do papiledema para normalidade papilar. A presença ou ausência de papiledema na HII pode estar relacionada ao período no qual seu diagnóstico é realizado. PALAVRAS-CHAVE: cefaléia, pressão intracraniana, papiledema, pseudotumor cerebral.

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Section: Discussionmentioning

confidence: 99%

“…The main abnormality disclosed by examination is papilledema, occasionally with hemorrhagic exsudates 13 . However, its absence does not exclude IIH 2,13,[18][19][20][21][22] .…”

Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

“…However, its absence does not exclude IIH 2,13,[18][19][20][21][22] . Paralysis of the abducent nerve, trochlear and facial paralysis 23 are also common findings.…”

Section: Discussionmentioning

confidence: 99%

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