Benign Subtentorial Supracollicular Cyst as a Cause of Obstructive Hydrocephalus

Alexander, Eben

doi:10.3171/jns.1953.10.3.0317

Cited by 40 publications

(5 citation statements)

References 11 publications

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“…Less frequent but also well recognized are cysts in the posterior fossa (Trowbridge and French, 1952;Alvord and Marcuse, 1962;Matson, 1969). Cysts around the base of the brain, and in the region of the subarachnoid cisterns are uncommon (Matson, 1969), although occasional reports have dealt with cysts in the chiasmatic region (Dott and Gillingham, 1958), in the quadrigeminal plate cistern (Alexander, 1953;Kruyff, 1965), and in the cisterna ambiens (Katagiri, 1960).…”

mentioning

confidence: 99%

Cerebral arachnoid cysts in children

Harrison

1971

Journal of Neurology, Neurosurgery & Psychiatry

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confidence: 99%

Cerebral arachnoid cysts in children

Harrison

1971

Journal of Neurology, Neurosurgery & Psychiatry

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“…Hamby and Gardner (1935) thought in their case that the cyst had formed from a ventricular diverticulum, but since their patient had suffered a fall they could not exclude trauma as a causative factor. Alexander's (1953) patient had had severe pertussis at the age of 5 months so the cyst could not necessarily be considered congenital in origin. In Dott and Gillingham's (1958) case there was a history of 'minor birth trauma' so that again trauma could not be excluded as the basic mechanism for the cyst formation.…”

Section: Discussionmentioning

confidence: 99%

“…Although the cyst wall was composed of ependymal cells there was neither radiological nor surgical evidence of a communication between the cyst and the ventricular system. Alexander (1953) reported an 18month-old patient with an arachnoid cyst of the quadrigeminal cistern. Severe obstructive hydrocephalus was relieved by the operative establishment of a communication between the posterior end of the third ventricle and the cyst.…”

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Radiological and Clinical Features of an Arachnoid Cyst of the Quadrigeminal Cistern

Lourie¹,

Berne²

1961

Journal of Neurology, Neurosurgery & Psychiatry

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Cummins, Taveras, and Schlesinger (1960) Case Report A 13-month-old boy was admitted to the Syracuse Memorial Hospital on March 29, 1960, because of regression in neuromuscular function and a rapidly enlarging head. The baby had developed normally to the age of 10 months at which time he could stand with help and spoke a few simple words. He then became irritable, slowly lost the ability to stand or sit, and at 12 months developed an internal squint of the right eye.The mother's pregnancy and delivery were uneventful. There was no history of infectious disease in childhood.Physical examination revealed a very irritable child. The skull was enlarged (52 5 cm. maximum circumference), but the scalp veins were flat and no cranial bruit was heard. Neurological examination demonstrated a right abducens palsy, bilateral papilloedema, a positive MacEwen's sign, and severe generalized spasticity and clonus. The blood count and urine analysis were indeterminate. Plain skull films showed widened sutures, an enlarged vault, and a simple linear fracture in the right parietal bone.On the second hospital day subdural taps were performed through the enlarged anterior fontanelle, but no fluid was obtained.Two days later contrast radiographs were obtained. (A detailed description of the pertinent films made during this and subsequent studies is given beneath Figs. I and 2.)A gas ventriculogram was performed first because of 374

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“…Wolf and Banford (1935), Van Wagenen and Air (1934) assumed that fluid is produced within Greenwood 1949, Alexander 1953, Huckmann et al 1970, Robinson 1971, Danzinger 1975). Wolf and Banford (1935), Van Wagenen and Air (1934) assumed that fluid is produced within Greenwood 1949, Alexander 1953, Huckmann et al 1970, Robinson 1971, Danzinger 1975).…”

Section: Pathologymentioning

confidence: 99%

Cavum vergae cyst as a cause of hydrocephalus, ?Almost Forgotten??

1986

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Cavum vergae cysts (CVC) are cerebral midline malformations, which usually have no clinical manifestations. In rare cases, however, non-communicating cysts can cause hydrocephalus by obstruction of the aqueduct. Initially, from 1930 to 1960 operative treatment consisted in open cysto-ventriculostomy, i.e. establishing a communication between CVC and the ventricles, via a transventricular approach. In the era of CT, symptomatic treatment of the hydrocephalus by ventriculo-atrial or ventriculo-peritoneal shunts is preferred. All the wellknown complications of shunting are encountered. Based on the experience of our cases of CVC with clinical manifestations, we here propose a definitive causal treatment which can be achieved by draining the cyst contents into the ventricles via a stereotactically introduced catheter.

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Benign Subtentorial Supracollicular Cyst as a Cause of Obstructive Hydrocephalus

Cited by 40 publications

References 11 publications

Cerebral arachnoid cysts in children

Cerebral arachnoid cysts in children

Radiological and Clinical Features of an Arachnoid Cyst of the Quadrigeminal Cistern

Cavum vergae cyst as a cause of hydrocephalus, ?Almost Forgotten??

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