�ber Ver�nderungen der Kreislaufdynamik des ruhenden Menschen unter Einwirkung von Ger�uschen

Lehmann, G.; Tamm, J.

doi:10.1007/bf00702457

Cited by 14 publications

(10 citation statements)

References 6 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Our study indicated a clear trend: decrease of SP and HR and increase of DP, and so are in agreement with the original studies of Lehmann et al ( 1956) and the studies of Kubik et al ( 1971), Paranko et al ( 1967) and Arkadjewskii ( 1966) The average changes in SP, DP and HR were not large; the findings in our study therefore do not conflict with observations by Jansen et al ( 1964) and Ettema et al ( 1969), who found SP, DP and HR to remain rather constant during noise exposure.…”

Section: Blood Pressure and Heart Ratesupporting

confidence: 94%

“…Ettema ( 1967) and Blohmke et al ( 1967) postulated a simultaneous increased tone of sympathicus and of parasympathicus with a kind of equilibrium maintained, under conditions of mental load Noise exposure appears to exert a similar effect: increased diastolic blood pressure, decreased sinus arrhythmia However for further clarification experiments with exposure to different types of noise and with examination of different parameters of response (e g neurophysiological data, perfusion of blood vessels) have to be carried out This new concept of noise effect is strengthened by the failure to observe a clear effect on heart rate (present study, Mosskov 1972, Jansen et al 1964, Lehmann et al 1956 Jansen et al ( 1964) observed a decreased peripheral blood volume, whereas heart rate and blood pressure remained rather at the same level; this observation was not interpreted by these authors as due to increased tone of sympathicus and parasympathicus with an equilibrium between both; this equilibrium will not always be complete, oscillations in both directions will occur, due to the characteristics of experimental conditions and of subjects exposed.…”

Section: Conflicting Datamentioning

confidence: 63%

See 1 more Smart Citation

II. Extra-auditory effects in short-term exposure to aircraft and traffic noise

Mosskov

Ettema

1977

Int. Arch Occup Environ Heath

View full text Add to dashboard Cite

Summary A series of human studies was performed in 12 young healthy male subjects to investigate extra-auditory effects of short-term ( 15 min) exposure to aircraft noise ( 84-91 d B(A)), to traffic noise (Leq 83 5 d B(A)), to mental load, and to combined load (noise + mental load) Exposure to noise caused increase of diastolic blood pressure and respiratory rate, decrease of pulse pressure and quotient of heart rate/respiratory rate and also decrease of performance in a mental task Aircarft and traffic noise generally induce similar effects, which go into the same direction as induced by mental performance alone Combined exposure induces a stronger (but not additive) effect on diastolic blood pressure and respiratory rate than each condition alone There is suggestive evidence that exposure to noise and performance of an information processing task both induce simultaneous increase of sympathic and parasympathic tone.

show abstract

Section: Blood Pressure and Heart Ratesupporting

confidence: 94%

Section: Conflicting Datamentioning

confidence: 63%

II. Extra-auditory effects in short-term exposure to aircraft and traffic noise

Mosskov

Ettema

1977

Int. Arch Occup Environ Heath

View full text Add to dashboard Cite

show abstract

“…Chronic stress including noise has been found to accelerate the development of structural vascular changes in the precapillary resistance vessels and also hypertension in animals (26). This and other studies have shown that shorttime exposure to noise causes a temporary increase in blood pressure (15)(16)(17)(18)(19)(20). Repeated exposure to noise could then explain the increased prevalence of hypertension observed in humans exposed to severe noise for many years (5-8).…”

Section: -822991mentioning

confidence: 59%

Circulatory Effects of Noise

Andrén

Hansson

Eggertsen

et al. 1983

Journal of Internal Medicine

View full text Add to dashboard Cite

Thirteen patients with mild essential hypertension, mean age 44 years (range 21–59), were studied during “stress” before and after postsynaptic α‐adrenoceptor blockade and combined postsynaptic α‐ and non‐selective β‐adrenoceptor blockade. Loud broad band noise (100 dBA for 10 min) was used as the stress stimulus. Exposure to noise caused a significant increase in systolic (7%, p<0.05), diastolic (9%, p<0.01) and mean arterial pressure (6%, p<0.01). The blood pressure elevation was caused by an increase in total peripheral resistance (12%, p<0.05). There was no significant change in heart rate, stroke volume or cardiac output. The blood pressure response during noise stimulation was not affected by postsynaptic α‐adrenoceptor blockade (prazosin, 2 mg orally). The hemodynamic reaction pattern, however, was totally reversed. Thus, the cardiac output increased significantly (9%, p<0.05), while the total peripheral resistance tended to decrease. Combined postsynaptic α‐ and non‐selective β‐adrenoceptor blockade (la‐be talol, 200 mg orally) inhibited the increase in systolic blood pressure caused by noise, while the diastolic and mean arterial pressures still increased significantly (5%, p<0.01). Labetalol effectively blocked the stress‐induced increase in total peripheral resistance and there was no significant increase in cardiac output after combined α‐ and β‐adrenoceptor blockade. Exposure to noise caused a significant increase in circulating noradrenaline (20%, p<0.05). Plasma adrenaline and plasma renin activity were not affected by noise stimulation. These results suggest that blood pressure elevation is essential during “stress” but that the hemodynamic pattern causing blood pressure elevation may vary and may be affected by pharmacological blockade of various parts of the sympathetic nervous system.

show abstract

“…[8][9][10] Test persons who are exposed to high noise levels have shown decreases in electrical skin resistance, skin temperature, and blood flow in peripheral blood vessels due to vasoconstriction, and increases in blood pressure and heart rate, indicating an arousal of the autonomic nervous system and the endocrine system. [11][12][13][14][15][16] In contrast to the acute noise effects observed at higher-namely occupational-noise levels, physiological effects from relatively low environmental noise levels primarily occur when the sound level disturbs cognitive functions, causes emotional reactions, or interferes with activities of the individual such as mental tasks, relaxation, or sleep. 7 17-20 According to the general stress model, activation of the sympathetic and the endocrine systems (stress indicators) is associated with changes in physiological functions and metabolism, including blood pressure, cardiac output, blood lipids, glucose, blood clotting factors, and other functions.…”

mentioning

confidence: 99%

Health status as a potential effect modifier of the relation between noise annoyance and incidence of ischaemic heart disease

Babisch

Ising²,

Gallacher³

2003

Occup Environ Med

View full text Add to dashboard Cite

Aims: Traffic noise is a psychosocial stressor. Epidemiological studies suggest chronic noise stress to be a risk factor for cardiovascular disorders. Methods: In a prospective cohort study, the association between annoyance and disturbances due to road traffic noise and the incidence of ischaemic heart disease (IHD) was studied in 3950 middle aged men. Results: Depending on the questionnaire item, non-significant odds ratios for IHD incidence ranging from 0.9 to 1.4 were found for the highly noise annoyed/disturbed subjects when compared with the less annoyed/disturbed subjects, over the six year follow up period. However, this relation was strongly modified by the prevalence of pre-existing chronic diseases. In subjects free of any chronic disease at the beginning of the follow up, significant odds ratios between 1.7 and 3.0 were seen. In the subgroup with chronic diseases no such noise effects were seen. This surprising result of no effect in the group of people with a potential risk, due to pre-existing health problems, may be because of the dilution of the true effect due to recall bias. Conclusions: Annoyance and disturbance due to road traffic noise is associated with a higher incidence of IHD. Prevalence of disease can be an important effect modifier of the relation between noise annoyance and health outcomes.

show abstract

�ber Ver�nderungen der Kreislaufdynamik des ruhenden Menschen unter Einwirkung von Ger�uschen

Cited by 14 publications

References 6 publications

II. Extra-auditory effects in short-term exposure to aircraft and traffic noise

II. Extra-auditory effects in short-term exposure to aircraft and traffic noise

Circulatory Effects of Noise

Health status as a potential effect modifier of the relation between noise annoyance and incidence of ischaemic heart disease

Contact Info

Product

Resources

About