Beyond postural tachycardia syndrome

Mustafa, Hossam; Robertson, David

doi:10.1136/jnnp.2009.180620

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Cited by 2 publications

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“…However, NET dysfunction is rare . Moreover, decreased (albeit slightly) innervation of the skin encourages the view that we have identified a partial sympathetic dysfunction …”

Section: Discussionsupporting

confidence: 71%

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Small‐fiber neuropathy with cardiac denervation in postural tachycardia syndrome

et al. 2014

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“…However, NET dysfunction is rare . Moreover, decreased (albeit slightly) innervation of the skin encourages the view that we have identified a partial sympathetic dysfunction …”

Section: Discussionsupporting

confidence: 71%

“…37 Moreover, decreased (albeit slightly) innervation of the skin encourages the view that we have identified a partial sympathetic dysfunction. [37][38][39][40] POTS is currently defined as a syndromic dysautonomia with a change in autonomic nervous system function. The exact causes of POTS are unknown.…”

Section: Discussionmentioning

confidence: 99%

Small‐fiber neuropathy with cardiac denervation in postural tachycardia syndrome

et al. 2014

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Orthostatische Hypotension

Schimpf¹,

Veltmann²,

Borggrefe³

2011

Herzschr. Elektrophys.

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Syncope is a frequent clinical event in the general population and occurs in up to every second patient during their lifetime. Reflex syncope is the most prevalent mechanism and is often triggered by orthostatic stress. Orthostatic hypotension (OH) represents a rare cause in young patients but is an important differential diagnosis in the aged. The Framingham study revealed an increase in the incidence of OH-triggered syncope from 5.7 events/1000 person-years at the age of 60-69 to 11.1 in men who are 70-79 years of age. OH often constitutes a chronic, debilitating illness with significant reduction in the quality of life. Important causes are volume loss, side effects of different vasoactive drugs, and neurodegenerative or secondary autonomic diseases following long-standing diabetes or amyloid disease. OH is difficult to treat. The therapeutic goal is to improve postural symptoms, standing time, and prevention of syncopal events. Drug therapy alone is never adequate. Because orthostatic stress varies during the day, a patient-tailored approach that emphasizes education and several general actions is recommended together with physical therapy and isometric exercise maneuvers. Moderate and severe cases require additional drug treatment to increase peripheral vascular resistance.

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Beyond postural tachycardia syndrome

Cited by 2 publications

References 17 publications

Small‐fiber neuropathy with cardiac denervation in postural tachycardia syndrome

Small‐fiber neuropathy with cardiac denervation in postural tachycardia syndrome

Orthostatische Hypotension

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