Bilateral abducens paresis following Campylobacter jejuni enteritis

Kruijk, R.A. van der; Lampe, A. S.; Endtz, Hubert P.

doi:10.1016/0163-4453(92)93136-e

Cited by 11 publications

(2 citation statements)

References 2 publications

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“…In Keane's large series of 125 patients with bilateral sixth nerve paresis, none had an unruptured aneurysm of the basilar artery (Keane, 1976). In addition, post-infectious bilateral abducens pareses were reported as well, probably because of autoimmunogenic aetiopathogenesis (vd Kruijk et al, 1992). Recently, Schroeder published the case of a young man with bilateral abducens paresis after the intake of ÔEcstasyÕ (Schroeder and Brieden, 2000).…”

Section: Figurementioning

confidence: 99%

HTLV‐I myelopathy − atypical features

Deshpande¹

2002

Euro J of Neurology

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Section: Figurementioning

confidence: 99%

HTLV‐I myelopathy − atypical features

Deshpande¹

2002

Euro J of Neurology

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“…In Keane's large series of 125 patients with bilateral sixth nerve paresis, none had an unruptured aneurysm of the basilar artery (Keane, 1976). In addition, post‐infectious bilateral abducens pareses were reported as well, probably because of autoimmunogenic aetiopathogenesis (vd Kruijk et al ., 1992). Recently, Schroeder published the case of a young man with bilateral abducens paresis after the intake of ‘Ecstasy’ (Schroeder and Brieden, 2000).…”

mentioning

confidence: 99%

Bilateral paresis of the abducens nerve: report of two cases

Gaul

Pfau

Huk

et al. 2002

Euro J of Neurology

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Serum anti‐GQ_1b IgG antibodies recognize surface epitopes on Campylobacter jejuni from patients with Miller Fisher syndrome

Jacobs

Endtz

Meché

et al. 1995

Annals of Neurology

136

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Three patients who had diarrhea prior to the development of Miller Fisher syndrome are presented. Campylobacter jejuni was isolated from stool specimens from all patients. High titers of anti-GQ1b IgG antibodies were demonstrated in the serum of these patients by enzyme-linked immunosorbent assay and thin-layer chromatography overlay. In enzyme-linked immunosorbent assay inhibition studies the anti-GQ1b IgG antibodies bound specifically to whole bacteria of the Miller Fisher syndrome-associated C. jejuni strains. The presence of anti-GQ1b IgG binding epitopes on the surface of the C. jejuni from the patients was not exclusively associated with a specific Penner serotype. It is suggested that anti-GQ1b antibodies are formed during the initial infection that elicits Miller Fisher syndrome. The cross-reactivity of anti-GQ1b IgG antibodies with surface epitopes on Miller Fisher syndrome-associated C. jejuni strains supports the hypothesis of molecular mimicry between bacteria and neural tissue.

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Bilateral abducens paresis following Campylobacter jejuni enteritis

Cited by 11 publications

References 2 publications

HTLV‐I myelopathy − atypical features

HTLV‐I myelopathy − atypical features

Bilateral paresis of the abducens nerve: report of two cases

Serum anti‐GQ_1b IgG antibodies recognize surface epitopes on Campylobacter jejuni from patients with Miller Fisher syndrome

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Bilateral abducens paresis following Campylobacter jejuni enteritis

Cited by 11 publications

References 2 publications

HTLV‐I myelopathy − atypical features

HTLV‐I myelopathy − atypical features

Bilateral paresis of the abducens nerve: report of two cases

Serum anti‐GQ1b IgG antibodies recognize surface epitopes on Campylobacter jejuni from patients with Miller Fisher syndrome

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Product

Resources

About

Serum anti‐GQ_1b IgG antibodies recognize surface epitopes on Campylobacter jejuni from patients with Miller Fisher syndrome