Bilateral Lesion of the Occipital Lobes With Retention of Macular as Distinct From Panoramic Vision: Clinical Record: Histo-Pathological Record

Bramwell, Byrom; Bolton, Joseph Shaw; Robinson, Wtilliam

doi:10.1093/brain/38.4.447

Cited by 12 publications

(3 citation statements)

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“…In the remarkable case of permanent loss of peripheral vision during acute nephritis and convulsions reported by Bramwell et al (1915), the cerebral lesions which were ischemic lay for the most part in the lateral occipital region bilaterally in the most distal middle cerebral distribution, and were possibly the result of hypotension or hypoxia during the seizures rather than representing the primary hypertensive visual disturbance.…”

Section: Ictal Deficits Othermentioning

confidence: 92%

Transient Paralytic Attacks of Obscure Nature: The Question of Non-Convulsive Seizure Paralysis

1978

Can. j. neurol. sci.

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Section: Ictal Deficits Othermentioning

confidence: 92%

Transient Paralytic Attacks of Obscure Nature: The Question of Non-Convulsive Seizure Paralysis

1978

Can. j. neurol. sci.

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“…This association was not considered in the early literature. 3There are at least 3 reported cases of extensive scotomas with macular sparing, in 2 of which colour vision was intact in the spared area (Peters, 1896;Bramwell et al, 1915), while in the third colour vision was lost (Forster, 1890). The latter had an extensive lesion of the occipital cortex, involving the lingual and fusiform gyri.…”

Section: The Scotomasmentioning

confidence: 99%

A Century of Cerebral Achromatopsia

Zeki

1990

Brain

423

205

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This review is an enquiry into why the early clinical evidence for a colour centre in the cerebral cortex of man was so successfully dismissed for the best part of a century. The imperfection of this evidence cannot be the reason, for the same evidence that was rejected earlier is accepted today. Instead, it was because the prevalent concepts of vision as a function, and of the role of the cerebral cortex in it, dominated facts and prevented acceptance of evidence showing a specialization for colour in the visual cortex. It was only after those concepts were overthrown by the demonstration of functional specialization in the visual cortex of the primate that the evidence for a colour centre in the human brain became acceptable. Today, our new knowledge of the colour areas and pathways in the primate brain allows us to give a more complete account of the pathophysiology of cerebral achromatopsia in man.

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“…3. The lesions involve the retrogeniculate portions of the visual system on both sides, either the optic radiations or the calcarine cortex or both.…”

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confidence: 99%

Cerebral Blindness

Bergman¹

1957

AMA Arch NeurPsych

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Cerebral blindness, often called "cortical" blindness, may be defined as bilateral absence of light perception (under the usual conditions of testing) caused by disease of the cerebral hemispheres. The lesions involve the retrogeniculate portions of the visual system on both sides, either the optic radiations or the calcarine cortex or both. For the purpose of this study, fulfillment of the following clinical criteria was required: 1. The patient was totally blind. 2. The eyes themselves (that is, media, retina, and optic nerve) were normal or showed only minor abnormality, incapable of causing blindness. 3. The pupillary light reactions were normal.4. There was other evidence of bilateral cerebral disorder, either at the time the blindness developed or in the past. The limitations of these criteria will be discussed later.This entity is well known, and many cases have been reported since the classical paper of F\l=o"\rsterin 1890.11 These reports have usually emphasized the pathology of this condition, and many concerned themselves with the denial of blindness (Anton's syn¬ drome) that frequently occurs. Although denial of blindness was once believed to be a specific dysfunction relating to the visual system. Redlich and Dorsey24 and, more recently, Weinstein and his associates33·34

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Bilateral Lesion of the Occipital Lobes With Retention of Macular as Distinct From Panoramic Vision: Clinical Record: Histo-Pathological Record

Cited by 12 publications

References 0 publications

Transient Paralytic Attacks of Obscure Nature: The Question of Non-Convulsive Seizure Paralysis

Transient Paralytic Attacks of Obscure Nature: The Question of Non-Convulsive Seizure Paralysis

A Century of Cerebral Achromatopsia

Cerebral Blindness

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