“…Many of the scoring systems used to predict and measureseverity are complex, cumber-some and inaccurate [3]. Some of them are: a) the Acute Physiology, Age and Chronic Health Evaluation (Apache II) [4,7,9,14]; b) Structured Interview of Reported Symptoms (SIRS) [4,9]; c) the Ranson score [4,7,9]; d) Bedside index for severity in AP (BISAP) (blood urea nitrogen >25mg/dl, impaired mental status, Systemic inflammatory response syndrome, age >60 years and pleural effusions) [2,3,15]; e) The Harmless Acute Pancreatitis Score (HAPS) (no rebound tenderness and/ or guarding, normal hematocrit and normal serum creatinine level) allows rapid identification of patients who present mild AP in 98% of cases; f) CT severity index (CTSI) based on local complications and percentage of pancreatic necrosis seen on a CT scan [2,4,5,17,18]. Various laboratory tests and biomarkers for predicting AP outcome have been described: a) elevated C-reactive protein (CRP) [1,4,13,14,19,20]; b) elevated hematocrit (Ht) [5,7,14,21,22]; and c) high serum creatinine, as a doubtful predictor of pancreatic necrosis [23,24].…”