2018
DOI: 10.4103/ijps.ijps_56_18
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Birth palsy in congenital varicella syndrome: A lesson in anatomy

Abstract: While brachial plexus palsy sustained due to birth trauma is well known, congenital palsies are decidedly rare. We report such a case caused by congenital varicella syndrome, with associated congenital Horner's syndrome and heterochromia iridis. The surprising juxtaposition of a classic upper plexus palsy and a Horner's syndrome raises points of interest. Similar reports in literature are reviewed, and the genesis of a very characteristic group of findings is discussed.

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“…Up to 25% of the infants born to women who contract varicella may become infected [4,5]. Approximately 2% of fetuses exposed to maternal varicella infection in the first 20 weeks (usually between 13 and 20 weeks) of pregnancy have features of congenital varicella syndrome [4,6,7].…”
Section: Discussionmentioning
confidence: 99%
See 1 more Smart Citation
“…Up to 25% of the infants born to women who contract varicella may become infected [4,5]. Approximately 2% of fetuses exposed to maternal varicella infection in the first 20 weeks (usually between 13 and 20 weeks) of pregnancy have features of congenital varicella syndrome [4,6,7].…”
Section: Discussionmentioning
confidence: 99%
“…Congenital varicella syndrome is characterized by a number of clinical manifestations: premature labor and small for gestational age; cutaneous lesions (e.g., scars in a dermatomal distribution and aplasia cutis congenita); CNS and peripheral nervous system abnormalities (e.g., microcephaly, hydrocephalus, cortical/cerebellar atrophy, mental retardation, facial nerve palsy, phrenic nerve palsy, recurrent laryngeal nerve palsy, bulbar palsy, brachial plexus palsy, and intracranial calcifications); ocular abnormalities (e.g., cataracts, nystagmus, microphthalmia, chorioretinitis, and optic atrophy); autonomic nervous system dysfunction (e.g., Horner syndrome, neurogenic bladder, dysphagia, and anal sphincter dysfunction); neuromuscular/orthopedic abnormalities (e.g., talipes equinovarus or calcaneovalgus deformity, hypoplasia/atrophy of the limb, rudimentary digit, hypoplasia of ribs, and scoliosis); gastrointestinal anomalies (e.g., duodenal stenosis, jejunoileal atresia, Meckel diverticulum, colonic atresia, colonic stricture, small left colon syndrome, and sigmoid atresia); and genitourinary anomalies (e.g., hydronephrosis, hydroureter, renal dysplasia, renal agenesis, and undescended testes) [6][7][8][9][10][11][12][13][14][15][16][17][18].…”
Section: Discussionmentioning
confidence: 99%