Blood lead levels and hypothalamic-pituitary-adrenal function in middle-aged individuals

Ngueta, Gérard; Verner, Marc-André; Fiocco, Alexandra J.; Lupien, Sonia J.; Plusquellec, Pierrich

doi:10.1016/j.envres.2017.10.032

Cited by 5 publications

(1 citation statement)

References 40 publications

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“…In smaller studies, decreased TSH and increased free T4 were observed at PbB of 3.10 and 7.3 µg/dL, respectively (Abdelouahab et al 2008;Dundar et al 2006). In a large, cross-sectional study, increased TPO antibodies were observed in women with PbBs >2.9 µg/dL, with a significant positive trend (p=0.008) for increased TSH; in men, there was no association (Nie et al 2017 (Braun et al 2014); enhanced salivary cortisol response to cold stress in children at PbB 1.1-6.2 µg/dL (Gump et al 2008); no association between PbB and basal cortisol levels or cortisol levels under stress (Ngueta et al 2018); no association between PbB and serum vitamin D in children at PbB means 4.94-6.54 µg/dL (Kemp et al 2007); decreased serum IGF-1 in children at PbB 5-9 µg/dL (Fleisch et al 2013); and no association between PbB and diabetes in children at mean PbB 4.08 µg/dL (Moon 2013).…”

Section: Ocularmentioning

confidence: 93%

Toxicological profile for lead

Abadin

Ashizawa²,

Stevens³

et al. 2020

142

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This toxicological profile is prepared in accordance with guidelines developed by the Agency for Toxic Substances and Disease Registry (ATSDR) and the Environmental Protection Agency (EPA). The original guidelines were published in the Federal Register on April 17, 1987. Each profile will be revised and republished as necessary. The ATSDR toxicological profile succinctly characterizes the toxicologic and adverse health effects information for these toxic substances described therein. Each peer-reviewed profile identifies and reviews the key literature that describes a substance's toxicologic properties. Other pertinent literature is also presented, but is described in less detail than the key studies. The profile is not intended to be an exhaustive document; however, more comprehensive sources of specialty information are referenced. CAS# 7439-92-1

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