“…Whether it is these changes in sleep architecture that impact on other symptoms or perhaps the transferability of some of the cognitive techniques employed (e.g., decatastrophizing techniques could equally be applied to symptoms of anxiety), however, remains to be seen. That said, there is some evidence for pre-post reductions in the discrepancy between subjective and objective sleep (Sforza et al, 2017), less activation in several brain mechanisms associated with attentional biases toward sleep-related cues (Nie & Dai, 2018), reductions in sleep-related dysfunctional cognitions (Chow et al, 2018), and reductions in sleep and wake time variability (Maurer et al, 2018) following CBT-I. However, one of the main challenges in determining the mechanisms of action behind CBT-I in this way is that it is difficult to disentangle the outcomes of CBT-I from its potential mechanisms of action.…”