Bulbuskontusionen durch Flaschendeckel

Viestenz, Arne; Kchle, M.

doi:10.1007/s003470100504

Cited by 7 publications

(6 citation statements)

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“…Unintentional eye injuries from bottles containing pressurised drinks were first reported in the early 1960s [11,12] and repeatedly thereafter [1,2,7,8,10,11,13,15,17]. Between 1.1% [16], 2% [9], 2.9% [4] and 3% [1] of all open globe injuries are related to this type; in this study we found a proportion of 2.4% of all open globe injuries being related to injuries from bottles containing carbonized drinks.…”

Section: Discussionmentioning

confidence: 99%

Open globe injuries induced by glass bottles containing carbonated drinks

Schrader

Gramer

2009

Graefes Arch Clin Exp Ophthalmol

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Section: Discussionmentioning

confidence: 99%

Open globe injuries induced by glass bottles containing carbonated drinks

Schrader

Gramer

2009

Graefes Arch Clin Exp Ophthalmol

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“…Unintentional eye injuries from bottles containing pressurized drinks are very common all over the word [1, 2, 3, 4, 5]. We have several ocular contusion registers that confirm the dimension of this phenomenon [1, 2].…”

Section: Introductionmentioning

confidence: 62%

“…The anterior segment is the part of the eye that is involved in a large number of cases, with the following morphological changes: corneal stroma edema, Descemet's folds, hyphema, iridodialysis, traumatic cataract, and vitreous prolapse [1, 5]. Fortunately, anterior segment contusion has a good functional prognosis [5].…”

Section: Introductionmentioning

confidence: 99%

Ultrabiomicroscopy Anterior Segment Evaluation of Ocular Contusive Trauma Caused by Pressurized Bottled Drink Caps: A Case Report

Esposito¹,

Rosati²,

Mannino³

et al. 2018

Case Rep Ophthalmol

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We report the case of a patient presented to the emergency department because of a contusive trauma from a pressurized bottled drink cap. During the visit, the patient indicated that he had been hit in his left eye by a cork while he was opening a sparkling wine bottle. He underwent a total ophthalmology examination. He had an important reduction of visual acuity, corneal swelling, Descemet’s folds, and hyphema. Therefore, we decided to perform ultrabiomicroscopy (UBM) of the anterior segment to study the endothelial damage and Descemet’s membrane. UBM images confirmed the direct biomicroscopy, highlighting the damaged location.

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“…Sports-related injuries are commonly implicated causative agents, but a variety of other causes have been reported, including airbags, elastic cords injuries, bottle caps and corks, airsoft pellets, and paintball guns. [10][11][12][13][14][15] Elevated IOP in blunt trauma can be secondary to angle recession, inflammation, intraocular bleeding, lenticular dislocation, and, in severe blunt trauma, phacoanaphylaxis secondary to capsular rupture. [16][17] Multivariate analysis of 6,021 cases in the US Eye Injury Registry identified key features associated with the development of traumatic glaucoma: advancing age, presenting visual acuities <6/60, iridial or lenticular injuries, hyphema, or angle recession.…”

Section: Discussionmentioning

confidence: 99%

Traumatic glaucoma with features of unilateral pigment dispersion

Bowler¹,

Ellul²,

Gouws³

2014

Digit J Ophthalmol

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SummaryWe report a patient with traumatic glaucoma with features of unilateral pigment dispersion. This rare form of secondary glaucoma has only been reported twice previously, with both patients demonstrating angle recession, indicating associated damage to the trabecular meshwork. To our knowledge, this is the first such case reported in which angle recession was absent. Case ReportA 68-year-old white man presented at Conquest Hospital, Hastings, with a complaint of redness and discomfort in the left eye 2 days after suffering blunt trauma with the tip of a pool cue. Medical history was remarkable for myopia previously treated with radial keratotomies. On examination, the left eye had diffusely injected conjunctiva with anterior chamber cell, traumatic mydriasis, and an intraocular pressure (IOP) of 20 mm Hg, with no gonioscopic evidence of angle recession. Radial keratotomy scars were visible in both eyes. Neither eye had features of pigment dispersion. Fundus examination revealed cup:disc ratios of 0.3 in both eyes, with no disc features suggestive of glaucoma. He was treated as a case of traumatic iritis and started on a tapering course of topical 0.1% dexamethasone every 2 hours.At 4 weeks' follow-up, the left eye was found to have an IOP of 70 mm Hg and a quiet anterior chamber. Although the left eye was physiologically shorter than the right eye, it was noted to have a visibly asymmetric, deeper anterior chamber on examination. This finding was confirmed by measurements obtained from the IOLMaster (Carl Zeiss Meditec, Jena, Germany): axial length 26.44 mm in the right eye and 25.84 mm in the left eye, anterior chamber depth 3.21 mm in the right eye and 3.45 mm in the left eye. There were no features of pigment dispersion. The elevated IOP was believed to be secondary to steroid therapy, which was consequently discontinued. He was started on topical latanoprost once daily and dorzolamide/timolol twice daily. This had the effect of reducing his IOP to 18 mm Hg. At 22 weeks' follow-up, his IOP had risen to 54 mm Hg, and he was noted to have pigmented cells in the anterior chamber for which he was restarted on topical dexamethasone 3 times daily. A Humphrey visual field 24-2 test was performed, revealing a mean deviation of −12.32 dB and an inferior arcuate scotoma. His IOP increased to 67 mm Hg, and he was started on oral acetazolamide 250 mg twice daily and referred to a glaucoma specialist.On examination of the left eye by a specialist 3 days later, the IOP was 42 mm Hg, the anterior chamber was asymmetrically deep with peripheral iris concavity, and there were midperipheral iris transillumination defects without other signs of iris damage. Gonioscopy showed evidence of Sampaolesi's line and no clinical evidence of angle recession (Figures 1-2). The cup:disc ratio in the left eye was 0.65, with superior neuroretinal rim thinning.The patient was diagnosed with traumatic pigment dispersion syndrome without angle recession and was started on maximal topical IOP reduction therapy; other medications were disco...

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Bulbuskontusionen durch Flaschendeckel

Abstract: Bottle cap injuries are not as harmless as supposed. Bottle caps may induce severe ocular damage due to the high-impact energy.

Cited by 7 publications

References 0 publications

Open globe injuries induced by glass bottles containing carbonated drinks

Open globe injuries induced by glass bottles containing carbonated drinks

Ultrabiomicroscopy Anterior Segment Evaluation of Ocular Contusive Trauma Caused by Pressurized Bottled Drink Caps: A Case Report

Traumatic glaucoma with features of unilateral pigment dispersion

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