Burn pathophysiology in man II. Sequential oxygen transport and acid-base alterations

Among 287 children with burns treated over a recent two-year period, 13 (5%) showed evidence of encephalopathy. The major clinical symptoms were an altered sensorium and seizures. The majority of symptoms began later than 48 hours after the burn and were accompanied by multiple metabolic aberrations including hypocalcemia. Three children had a relapsing course, and 1 had temporarily enlarged cerebral ventricles. Eleven children improved to normal. In the majority of instances, burn encephalopathy probably reflects central nervous system dysfunction resulting from complex metabolic, hematological, and hemodynamic abnormalities rather than from a single metabolic abnormality.

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Burn encephalopathy in children

Mohnot

Snead

Benton

1982

Annals of Neurology

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Burn shock in untreated and saline-resuscitated guinea pigs development of a model

Wolfe

Miller

1976

Journal of Surgical Research

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Method for estimation of the perfusion defect in shock

Shoemaker¹,

Launder²,

Castagna³

et al. 1976

Journal of Surgical Research

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Burn pathophysiology in man II. Sequential oxygen transport and acid-base alterations

Cited by 4 publications

References 10 publications

Burn encephalopathy in children

Burn encephalopathy in children

Burn shock in untreated and saline-resuscitated guinea pigs development of a model

Method for estimation of the perfusion defect in shock

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