2009
DOI: 10.1016/j.jelectrocard.2008.12.019
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Cardiac imaging in right ventricular cardiomyopathy/dysplasia—how does cardiac imaging assist in understanding the morphologic, functional, and electrical changes of the heart in this disease?

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Cited by 6 publications
(6 citation statements)
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“…It is likely that the limited parameters included in the TFC do not fully encompass and reflect the intricacies of RV structure and function . This is especially true in a disease such as ARVC/D where phenotype expression is quite variable, and regional changes in structure and function are common. Indeed, applying a limited number of fixed criteria to try to diagnose a morphologically heterogeneous disease can be troublesome and may lead to difficulties differentiating the RV in this pathological condition from the physiological changes of the RV associated with exercise.…”
Section: Discussionmentioning
confidence: 99%
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“…It is likely that the limited parameters included in the TFC do not fully encompass and reflect the intricacies of RV structure and function . This is especially true in a disease such as ARVC/D where phenotype expression is quite variable, and regional changes in structure and function are common. Indeed, applying a limited number of fixed criteria to try to diagnose a morphologically heterogeneous disease can be troublesome and may lead to difficulties differentiating the RV in this pathological condition from the physiological changes of the RV associated with exercise.…”
Section: Discussionmentioning
confidence: 99%
“…The TFC rely on only two RV anatomical measures (RV outflow tract diameter from the parasternal long‐axis view [PLAX RVOT] and short‐axis view [SAX RVOT]), and only one functional measure (RV fractional area change [RV FAC]) . These parameters may not reflect enough nuance to accurately discriminate the complexities of RV structure and function, especially in a condition such as ARVC/D where there is wide variability in phenotype expression …”
Section: Introductionmentioning
confidence: 99%
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“…The prevalence of ARVC in the general population has been reported to be 1:5000, affecting men more frequently than women with a ratio of 3:1 ( 5 , 6 ). The accuracy of available prevalence data is, however, still debated largely due to the complexities in diagnosing the disease ( 7 ). Currently, the diagnosis of ARVC can be established using a set of major and minor criteria proposed by an International Task Force in 1994 and updated/revised in 2010 ( 8 , 9 ).…”
Section: Introductionmentioning
confidence: 99%
“…Although the recent task force revision has improved specificity of diagnosis, there has been little impact upon diagnostic sensitivity ( 3 ) which may be due to the reliance on only two RV anatomical measures (the RV outflow tract from a parasternal long (RVOT PLAX ) and short axis (RVOT 1 )) and one functional measure (RV fractional area change (RVFAC)). It is likely that these parameters do not fully reflect the complexities of RV structure and function specifically in ARVC where phenotype expression is variable ( 1 , 7 ) and regional changes in structure and function are likely. A more comprehensive echocardiographic assessment of the RV in ARVC patients is warranted ( 10 ) in order to potentially improve diagnostic accuracy.…”
Section: Introductionmentioning
confidence: 99%