1998
DOI: 10.1006/clin.1998.4527
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Cell Death and Oxidative Damage in Inflammatory Myopathies

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Cited by 72 publications
(57 citation statements)
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“…Altered reduced/oxidized markers has been reported in MD patients and animal models (7,8,13,81). ROS produced in mdx mice and Col6a1 Ϫ/Ϫ mice might contribute to muscle pathology (80).…”
Section: Discussionmentioning
confidence: 99%
“…Altered reduced/oxidized markers has been reported in MD patients and animal models (7,8,13,81). ROS produced in mdx mice and Col6a1 Ϫ/Ϫ mice might contribute to muscle pathology (80).…”
Section: Discussionmentioning
confidence: 99%
“…Previous studies [17][18][19][20] have demonstrated iNOS overexpression in skeletal muscles of chronic heart failure patients and animals. Such iNOS overexpression has been shown to be induced by inflammatory cytokines and the priming effect of gamma interferone (γ-IFN) in chronic heart failure [21], iNOS overexpression has also been demonstrated in skeletal muscles of autoimmune inflammatory myopathies [22]. NOS activity inhibits mitochondrial respiration in skeletal muscles [23,24].…”
Section: Discussionmentioning
confidence: 99%
“…NO also mediates denervation responses, inflammatory myopathy, aging, and neuromuscular transmission (Tews et al, 1997a,b;Capanni et al, 1998;Ribera et al, 1998;Tews and Goebel, 1998). Therefore, the collective effects of NO have a significant impact on muscle pathophysiology.…”
Section: Discussionmentioning
confidence: 99%