1962
DOI: 10.3171/jns.1962.19.5.0419
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Cerebral Swelling and Ventricular Alterations Following Evacuation of Intracranial Extracerebral Hematoma

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Cited by 23 publications
(6 citation statements)
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“…Many pathological events may contribute to the development of an ICH after the evacuation of a chronic subdural hematoma. For example, damage to the cerebral vasculature secondary to perioperative parenchymal shift, a sudden increase in blood flow combined with defective vascular autoregulation, and hemorrhage into a previously undetected contusion have been proposed to explain the occurrence of delayed intracranial hematomas or SDH1,2,7). This mechanism is supported by several pathologic findings.…”
Section: Discussionmentioning
confidence: 95%
“…Many pathological events may contribute to the development of an ICH after the evacuation of a chronic subdural hematoma. For example, damage to the cerebral vasculature secondary to perioperative parenchymal shift, a sudden increase in blood flow combined with defective vascular autoregulation, and hemorrhage into a previously undetected contusion have been proposed to explain the occurrence of delayed intracranial hematomas or SDH1,2,7). This mechanism is supported by several pathologic findings.…”
Section: Discussionmentioning
confidence: 95%
“…aging of the cerebral vascular tree may not tolerate a sudden restoration of normal perfusion pressure in areas of deranged cerebral vascular autoregulation (5,10). The second hypothesis was suggested by Savadekar and Salunke.…”
Section: Discussionmentioning
confidence: 99%
“…[2,3] The azygous ACA may be associated with malformations like holoprosencephaly, agenesis of corpus callosum, meningoceles, lipoma, arachnoid cyst, and porencephalic cyst. [4][5][6][7] In the available literature, the association of an azygous ACA with a coexisting suprasellar tumor has not been described. The association of an azygous ACA with an anterior cranial fossa meningioma, as seen in our patient, may be coincidental.…”
Section: Sirmentioning
confidence: 99%
“…[6] Damage to the cerebral vasculature secondary to a perioperative parenchymal shift, a sudden increase in the blood flow combined with a defective vascular autoregulation, and hemorrhage into a previously undetected contusion have also been proposed to explain the occurrence of a delayed intracerebral hematoma or a SDH. [7,8] Long-term use of antiplatelet agents may lead to an altered coagulation state and precipitate a remote hemorrhage. [9] A slow evacuation of the hematoma is, therefore, of paramount importance and can reduce the incidence of this catastrophic complication.…”
Section: Sirmentioning
confidence: 99%