2012
DOI: 10.1007/s10571-012-9802-x
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Changes of Ribosomal Protein S3 Immunoreactivity and Its New Expression in Microglia in the Mice Hippocampus After Lipopolysaccharide Treatment

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Cited by 3 publications
(5 citation statements)
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References 50 publications
(57 reference statements)
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“…AMI treatment up-regulated Riok1 in all tissues studied, and pertinent to the remedial activities of AMI, it has been recently demonstrated that this kinase is a critical controller of AKTmediated cell survival (78). The consistent AMI-mediated reduction of Rps3a may also indicate that this is linked to potential cell protection mechanisms (79) and anti-inflammatory behavior (80). In addition to these highly conserved AMIresponse transcripts, many other factors consistently controlled across the brain are strongly implicated in potentially neuroprotective functions.…”
Section: Discussionmentioning
confidence: 94%
“…AMI treatment up-regulated Riok1 in all tissues studied, and pertinent to the remedial activities of AMI, it has been recently demonstrated that this kinase is a critical controller of AKTmediated cell survival (78). The consistent AMI-mediated reduction of Rps3a may also indicate that this is linked to potential cell protection mechanisms (79) and anti-inflammatory behavior (80). In addition to these highly conserved AMIresponse transcripts, many other factors consistently controlled across the brain are strongly implicated in potentially neuroprotective functions.…”
Section: Discussionmentioning
confidence: 94%
“…Many previous studies have focused on effects of LPS on neuronal degeneration and immune response in various regions of the brain [ 19 , 20 , 21 ]. In addition, our previous studies showed that systemic administration of 1 mg/kg LPS led to microglial activation without any neuronal damage as well as changes of DNA repair ability in the mouse hippocampus [ 6 , 11 ]. However, there is no study on LPS-induced changes of CB protein expression in the hippocampus.…”
mentioning
confidence: 99%
“…According to the method of our previous study [ 6 , 11 ], immunohistochemical staining for CB was performed using rabbit anti-CB (1:200, Millipore, Temecula, CA, U.S.A.), biotinylated goat anti-rabbit IgG (1:200, Vector, Burlingame, CA, U.S.A.) and streptavidin peroxidase complex (1:200, Vector). In order to establish the specificity of the immunostaining, a negative control test was carried out with pre-immune serum instead of primary antibody.…”
mentioning
confidence: 99%
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