Characteristics of culprit atheromatous plaques obtained in vivo by intravascular ultrasound radiofrequency analysis: Results from the CULPLAC study

Tissue characterization and phenotype classification in patients presenting with acute myocardial infarction: Insights from the iWonder study

“…Finally, this was the first study proposing a phenotype classification for atherosclerotic lesions in patients with ACS using iMAP‐IVUS. Previous studies have used radiofrequency IVUS derived from a 20 MHz IVUS catheter and applying the proprietary analysis software called VH‐IVUS . Available data with iMAP‐IVUS were limited to describing plaque composition, without the proposition of an algorithm for phenotype classification .…”

Evolution of nonculprit coronary atherosclerotic plaques assessed by serial virtual histology intravascular ultrasound in patients with ST-segment elevation myocardial infarction and chronic total occlusion

“…In these studies, it was generally accepted that TCFA with abundant NC was inclined to be ruptured, a so-called vulnerable plaque. Hong et al 17 and Cascon-Perez et al 18 reported that TCFA was more frequent in patients with acute coronary syndrome (ACS) than in those with stable angina, and the culprit lesions of ACS had greater amounts of NC and smaller amounts of FF compared with the target lesion of stable angina. Nakamura et al 19 analyzed the nonculprit lesion between stable and unstable angina patients, where unstable angina patients presented with a higher prevalence of VH-TCFA than stable angina patients.…”

Caracterização Morfológica e Tecidual de Lesões Culpadas em Pacientes com Infarto Agudo do Miocárdio com Supradesnivelamento do Segmento ST Após Uso de Fibrinolítico. Análise com Ultrassom Intracoronário e Tecnologia iMAP®

“…16,18 One of the most often studied findings associated with plaque vulnerability is arterial remodeling. Initially described by Glagov et al 19 in 1987, the positive arterial remodeling has been observed in atherosclerotic plaques responsible for acute coronary events, 6,20,21 and is associated with the increase in CK-MB after PCI, 22 no-reflow phenomena during primary PCI, 23 recurrent ischemia after PCI, 24 major cardiovascular events in patients with unstable angina undergoing any form of revascularization, 25 and intimal hyperplasia after PCI with bare-metal 26 and drug-eluting stents. 27 In the present study, the mean arterial-remodeling index was 1.4 ± 1.0, greater than 1.05, thus characterizing the predominance of positive arterial remodeling and corroborating the aforementioned literature.…”