“…The basic patho-physiology involves the release of vasoconstrictive agents, endothelial damage, hyperpermeability of the capillaries and microangiopathic hemolysis [ 3 , 6 ]. Ascites is reported antenatally, after normal delivery and after cesarean section in the postpartum period, as well as during cesarean section in women with preeclampsia [ [4] , [5] , [6] , 8 , 9 ]. There are case reports of postpartum ascites caused by bladder rupture following vaginal delivery [ 10 ], tuberculosis peritonitis [ 11 ], idiopathic chylous ascites [ 12 ], pregnancy specific liver disease [ 13 ], lupus peritonitis [ 14 ], etc.…”