Although chest pain in association with ST-segment P atients who report chest pain receive priority attention from emergency department (ED) providers-especially when that pain is associated with electrocardiographic (ECG) changes and with cardiac biomarkers consistent with acute coronary syndrome (ACS). In these cases, a consulting cardiologist needs to quickly decide whether to transfer the patient for invasive heart catheterization. We report a challenging ED clinical situation in which cardiac injury and "acute abdomen" occurred simultaneously. Despite time constraints, a thorough and comprehensive patient evaluation must be performed in order to avoid unnecessary invasive interventions.
Case ReportIn July 2012, a 63-year-old woman presented at our emergency room with abdominal pain that had started several hours earlier. An abdominal computed tomogram showed signs of colonic thickening, thereby raising suspicion of ischemic colitis. As evaluated by a surgeon, the patient's clinical status (no guarding or abdominal rebounding), hemodynamic results, and laboratory values (normal lactic acid) did not warrant emergent surgical management. Therefore, the patient was at first treated in the ED via supportive measures that included pain management and the administration of fluids and antibiotics. Six to seven hours later, she reported chest pain. Cardiac enzyme tests, including retroactive measurements of troponin T from the first laboratory draw, showed abnormal levels of troponin T, rising from 0.13 to 0.23 ng/mL (negative <0.03 ng/mL), together with a normal creatine kinase level and an abnormal creatine kinase-MB fraction at 10.5 ng/mL (normal, <5 ng/mL). An ECG showed a 1-to 2-mm ST elevation in leads V 2 and V 3 , which was sufficiently suspect to trigger an urgent cardiology consultation to evaluate the possible need for cardiac catheterization in advance of abdominal surgery.Laboratory data showed a white-cell count of 8.5 ×10 9 /L, a hematocrit of 36.5%, and a platelet count of 191 ×10 9 /L. Two serial measurements of lactic acid were normal. Abdominal computed tomography with contrast agent revealed mildly dilated fluid-filled loops of small bowels, possible small-bowel obstruction secondary to adhesions, and a small area of nondistended small bowel between 2 distended loops.At our bedside evaluation, the patient was very uncomfortable because of diffuse severe pain in the mid-epigastric and lower sternal areas, associated with diaphoresis and nausea. Vital signs were a temperature of 98.3 °F, a heart rate of 90 beats/min, a respiratory rate of 18 breaths/min, an oxygen saturation of 98% on 2 L/min oxygen