Chest ultrasound for diagnosis of transfusion-related acute lung injury

Cacho, B. Rosich del; Ortiz, Claudia; Rodríguez-Fanjul, Javier

doi:10.1016/j.medine.2020.01.001

Cited by 1 publication

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“…46,47 A bilateral irregular reticulonodular pattern 53 or pleural effusion can also be seen. 54 In people, TRALI can manifest as heterogenous parenchymal consolidations with air bronchograms, and ground-glass appearance of the lungs is sometimes seen on CT. 54 Thoracic ultrasound detected bilateral coalescent B-lines, pleural thickening, and subpleural consolidations in case reports in people with TRALI, 55,56 consistent with findings in people with ARDS. 41 In some cases, lung ultrasonographic changes can be present before significant changes can be detected on thoracic radiographs.…”

Section: Thoracic Imagingsupporting

confidence: 65%

“…Thoracic ultrasound detected bilateral coalescent B‐lines, pleural thickening, and subpleural consolidations in case reports in people with TRALI, 55,56 consistent with findings in people with ARDS 41 . In some cases, lung ultrasonographic changes can be present before significant changes can be detected on thoracic radiographs 55 …”

Section: Transfusion‐related Acute Lung Injurysupporting

confidence: 61%

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Noncardiogenic pulmonary edema in small animals

Unger

Martin

2023

J Vet Emergen Crit Care

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Objective To review various types of noncardiogenic pulmonary edema (NCPE) in cats and dogs. Etiology NCPE is an abnormal fluid accumulation in the lung interstitium or alveoli that is not caused by cardiogenic causes or fluid overload. It can be due to changes in vascular permeability, hydrostatic pressure in the pulmonary vasculature, or a combination thereof. Possible causes include inflammatory states within the lung or in remote tissues (acute respiratory distress syndrome [ARDS]), airway obstruction (post‐obstructive pulmonary edema), neurologic disease such as head trauma or seizures (neurogenic pulmonary edema), electrocution, after re‐expansion of a collapsed lung or after drowning. Diagnosis Diagnosis of NCPE is generally based on history, physical examination, and diagnostic imaging. Radiographic findings suggestive of NCPE are interstitial to alveolar pulmonary opacities in the absence of signs of left‐sided congestive heart failure or fluid overload such as cardiomegaly or congested pulmonary veins. Computed tomography and edema fluid analysis may aid in the diagnosis, while some forms of NCPE require additional findings to reach a diagnosis. Therapy The goal of therapy for all types of NCPE is to preserve tissue oxygenation and reduce the work of breathing. This may be achieved by removing the inciting cause (eg, airway obstruction) and cage rest in mild cases and supplemental oxygen in moderate cases and may require mechanical ventilation in severe cases. Prognosis Prognosis is generally good for most causes of veterinary NCPE except for ARDS, although data are scarce for some etiologies of NCPE.

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