Cholelithiasis in infants: association with total parenteral nutrition and furosemide.

Callahan, Jean-Christophe; Haller, Jack O.; Cacciarelli, Alexander; Slovis, Thomas L.; Friedman, Abraham

doi:10.1148/radiology.143.2.6803294

Cited by 31 publications

(11 citation statements)

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“…Infection is a likely cause since both of therm were suspected of harboring sepsis at different stages of their management. Another likely cause is the combination of total intravenous nutrition and furosemide which has been suspected of causing cholelithiasis in premature infants [8,9]. Both our patients were on this combination for an extended period of time.…”

Section: Discussionmentioning

confidence: 80%

Transient gallbladder distention in sick premature infants: the value of ultrasonography and radionuclide scintigraphy

El-Shafie

Mah

1986

Pediatr Radiol

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Transient gallbladder distention in sick premature infants has been reported with increasing frequency over the last few years. It is important to be aware of this entity of gallbladder disease which resolves spontaneously and to be able to differentiate it from other problems that require surgical correction. We believe that ultrasonography is most helpful in confirming the diagnosis, excluding stone formation, and documenting any changes in size or appearance. However, there are no reliable ultrasonic criteria for distinguishing inflamed or pathologically distended gallbladders from noninflamed or temporarily enlarged gallbladders. Radioisotope imaging, though useful in demonstrating malfunction of the hepatobiliary system, should be interpreted with caution. In the final analysis, the dilemma as to whether gallbladder distention is a transient or persistent malfunction can be settled best by thoughtful clinical judgement.

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Section: Discussionmentioning

confidence: 80%

Transient gallbladder distention in sick premature infants: the value of ultrasonography and radionuclide scintigraphy

El-Shafie

Mah

1986

Pediatr Radiol

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“…Cholelithiasis, once considered to be rare in preterm infants [7], is encountered more often nowadays especially in units where abdominal ultrasonography is performed routinely. Gallstones in infants are postulated to result from immature physiological regulation of bile salt secretion which is exaggerated by hepatotoxicity of TPN or prolonged fasting [8].…”

Section: Discussionmentioning

confidence: 99%

Nephrocalcinosis, cholelithiasis, and umbilical vein calcification in a premature infant

et al. 1994

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A case of calcification of the umbilical vein in a premature infant with nephrocalcinosis and cholelithiasis is described. Such an association might have resulted from prematurity, bronchopulmonary dysplasia, umbilical vein catheterization, total parenteral nutrition, and furosemide therapy. Follow-up ultrasound examination at 9 months of age revealed spontaneous resolution of calcification of both the gallbladder and the umbilical vein but not of the kidneys.

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“…Situations responsible for gallbladder stasis such as dehydration (Callahan et al, 1982) fasting (Bolondi et al, 1985) or total parenteral nutrition (Roslyn et al, 1983;Messing et al, 1983) are known to further biliary precipitations. Of course, one cannot exclude the presence of these factors in Schaad's and Heim-Duthoy's studies.…”

Section: Discussionmentioning

confidence: 99%

Incidence of gallbladder lithiasis after ceftriaxone treatment

Cometta¹,

S²,

Iten³

et al. 1990

J Antimicrob Chemother

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Ceftriaxone has potent activity against a broad range of Gram-positive and Gramnegative bacteria. While it is Hjminat^ mainly by the kidney, 10-20% of the drug is eliminated in the bile and ceftriaxone salt precipitates have been described in the gallbladder of animals dosed with ceftriaxone. The purpose of the present study was to investigate the incidence of binary litniasis 6 and 12 months after treatment with ceftriaxone and to compare it with that in patients treated with amoxyciUin/ clavulanate. Biliary ultrasonography was performed at the start of treatment, at 6 months and at 12 months after the beginning of the study. One hundred patients were randomized and 74 were evaluable: 34 were given amoxycillin/clavulanate, 40 ceftriaxone. Gallbladder litniasis developed in one patient 12 months after the amoxycillin/clavulanate treatment and in none in the ceftriaxone treatment arm. Biliary precipitate during ceftriaxone treatment was not looked for because this phenomenon was not known at the beginning of the study, but gallbladder precipitation that was seen in two patients given ceftriaxone during and at the end of treatment, respectively, resolved spontaneously.In conclusion, ceftriaxone treatment does not appear to lead to gallstone formation more often than an antibiotic that is not eliminated through the bile.

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Cholelithiasis in infants: association with total parenteral nutrition and furosemide.

Cited by 31 publications

References 0 publications

Transient gallbladder distention in sick premature infants: the value of ultrasonography and radionuclide scintigraphy

Transient gallbladder distention in sick premature infants: the value of ultrasonography and radionuclide scintigraphy

Nephrocalcinosis, cholelithiasis, and umbilical vein calcification in a premature infant

Incidence of gallbladder lithiasis after ceftriaxone treatment

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