Chronic Low-Back Pain Modulation Is Enhanced by Hypnotic Analgesic Suggestion by Recruiting an Emotional Network:<i>A PET Imaging Study</i>

Nusbaum, Fanny; Redouté, Jérôme; Bars, Didier Le; Volckmann, P.; Simon, François; Hannoun, Salem; Ribes, G; Gaucher, Jacques; Laurent, Bernard; Sappey-Marinier, Dominique

doi:10.1080/00207144.2011.522874

Cited by 41 publications

(33 citation statements)

References 33 publications

(44 reference statements)

Supporting

Mentioning

Contrasting

Order By: Relevance

“…These studies have concluded that hypnosis can impact pain by affecting a number of different neurophysiological processes that make up the pain matrix, rather than by influencing a single mechanism or process. Specifically, research indicates that hypnosis can impact activity in: (1) the periphery and spinal cord [67][68][69], (2) the thalamus [65,70,71], (3) the sensory cortices [72,73], (4) the insula [70,71,73], (5) the ACC [35,70,71,[73][74][75], and (6) the prefrontal cortex [70,71,73,76]. Given the extensiveness of these previous reviews, this section will briefly review past research that has examined the neurophysiological effects of hypnotic analgesia, specifically focused on the role of suggestion type [35,72,77].…”

Section: Neurophysiological Effects Of Hypnosismentioning

confidence: 99%

“…Given the extensiveness of these previous reviews, this section will briefly review past research that has examined the neurophysiological effects of hypnotic analgesia, specifically focused on the role of suggestion type [35,72,77]. Additionally, three studies that were not included in these previous reviews will be discussed, including one recent study evaluating the neurophysiological mechanisms associated with hypnotic induction and two studies that studied the neural processes correlated with suggestions [73,76,78].…”

Section: Neurophysiological Effects Of Hypnosismentioning

confidence: 99%

“…Another recent study by Nusbaum and colleagues [73] in patients with chronic back pain evaluated the neural networks activated during normal alertness and hypnosis for both direct analgesic suggestions (i.e., to alter the intensity and location of pain) and indirect suggestions (i.e., referencing general well-being with no mention of the pain). Using positron emission tomography, participants were given either direct or indirect suggestions first during normal alertness and then followed by a hypnotic induction.…”

Section: Neurophysiological Effects Of Hypnosismentioning

confidence: 99%

“…Studies have made beginning steps in pursuing this type of research [35,72,73,77]. As we learn more about the pain mechanisms that are specific to an individual's pain experience, we may be able to test specific suggestions that target those neural pathways [34].…”

Section: Research Implicationsmentioning

confidence: 99%

See 3 more Smart Citations

Neurophysiology of pain and hypnosis for chronic pain

Dillworth

Mendoza

Jensen

2011

Behav. Med. Pract. Policy Res.

View full text Add to dashboard Cite

In the past decade there has been a dramatic increase in (1) understanding the neurophysiological components of the pain experiences, (2) randomized clinical trials testing the efficacy of hypnotic treatments on chronic pain, and (3) laboratory research examining the effects of hypnosis on the neurophysiological processes implicated in pain. Work done in these areas has not only demonstrated the efficacy of hypnosis for treating chronic pain but is beginning to shed light on neurophysiological processes that may play a role in its effectiveness. This paper reviews a selection of published studies from these areas of research, focusing on recent findings that have the most potential to inform both clinical work and research in this area. The paper concludes with research and clinical recommendations for maximizing treatment efficacy based on the research findings that are available. KEYWORDSHypnosis, Hypnotic analgesia, Chronic pain, Neurophysiology, Hypnotic suggestions Chronic pain, defined as pain lasting longer than 6 months [1], is a complex experience that requires multifaceted approaches for both evaluation and treatment [1,2]. Chronic pain is considered to have an underlying biological basis, for which medications and physical treatments are commonly prescribed. Medical approaches for pain relief and management include pharmacological and surgical interventions as well as physical therapy. While nonsteroidal antiinflammatory drugs are commonly used to treat mild pain from inflammation, opioid analgesics are the mainstay of pharmacologic treatment of moderate to severe pain [2][3][4][5][6]. However, prolonged use of opioids may result in opioid tolerance and opioid-induced hyperalgesia, which is an increased sensitivity to pain. These problems, coupled with unpleasant medication side effects, may lead to discontinuation of treatment [7]. Depending on the patient's condition and cause of pain, surgery is typically considered for the treatment of chronic pain when it is deemed medically necessary or after other treatments have failed. These include intrathecal drug delivery [8], spinal cord stimulation [9], radiofrequency ablation [10], and chemical sympathectomy [11]. One concern is that even though surgical procedures can provide pain relief, they also may permanently damage the person's ability to perceive other sensations, such as light touch and temperature changes, and can cause different pain problems to occur.Medical approaches for pain reduction are only one element of an interdisciplinary approach, since gains in physical and social functioning are important goals as well [12]. Psychosocial and neuropsychological models of chronic pain show that pain treatment cannot focus solely on the sensation of pain itself (e.g., pain intensity) but must also consider the affective (e.g., emotional suffering), cognitive (e.g., beliefs about pain) and behavioral (e.g., inactivity) responses which also impact the overall experience of pain [13][14][15]. This idea has been further supported by research ev...

show abstract

Section: Neurophysiological Effects Of Hypnosismentioning

confidence: 99%

Section: Neurophysiological Effects Of Hypnosismentioning

confidence: 99%

Section: Neurophysiological Effects Of Hypnosismentioning

confidence: 99%

Section: Research Implicationsmentioning

confidence: 99%

See 2 more Smart Citations

Neurophysiology of pain and hypnosis for chronic pain

Dillworth

Mendoza

Jensen

2011

Behav. Med. Pract. Policy Res.

View full text Add to dashboard Cite

show abstract

“…Combined with local anesthesia, beneficial effects of hypnosis on patients undergoing surgery have been previously described [8,9]. From an Ericksonian perspective "indirect" suggestions are theoretically approached as suggestions which can circumvent the censorship of consciousness to reach the "unconscious" where they can activate dormant potentials [8,10].…”

Section: Case Reportmentioning

confidence: 99%

Hypnosis and Transversus Abdominis Plane Block for Brugada Syndrome Anesthesia: A Case Report

Santos¹,

Cailleau²,

Coscoy³

et al. 2017

Clin Med Rev Case Rep

View full text Add to dashboard Cite

SummaryBrugada syndrome is a distinct form of an arrhythmic disease affecting ionic channels of the heart. We report the anesthetic management of a patient with Brugada syndrome for inguinal hernia repair which was managed satisfactorily with combined light anesthesia, hypnosis with indirect suggestions and transversus abdominis plane block. KeywordsAnesthesia, Brugada syndrome, Hypnosis, Transversus abdominis plane block found a Brugada syndrome but there was no history of angina, syncope and the effort tolerance was about eight Metabolic Equivalents (METS). Family history was negative for Brugada syndrome and for sudden cardiac death. The patient had no history of abnormal clinical bleeding and was not taking any medications. His ASA physical status classification was 2.Brugada syndrome was diagnostic in 2014 because of a chest pain during night. His investigations were within normal limit except surface electrocardiogram that found ST segments < 1 mm with saddle-type pattern (Brugada type 2). A provocative drug test with intravenous administration of ajmaline induced type 1 ECG morphology with ST-segment elevation > 2 mm).Because he has a Brugada syndrome type 2 with no history of ventricular fibrillation or syncope and no family history of sudden cardiac death, cardioverter defibrillator was not implanted [1][2][3].Monitoring consisted of ECG with continuous ST segment analysis, a pulse oximeter and non-invasive blood pressure measurement. He was connected to Oxygen 2 L/min with nasal cannula and an external defibrillator pads with an external defibrillator was placed. Venous puncture (18G) was performed. After premedication with midazolam (1 mg), a TAP block was performed with 1% lidocaine adrenaline with ultrasound guidance and a total dose of 200 mg was injected. In addition to that, anesthetists nurse started hypnosis giving permissive and indirect suggestions of well-being. Even though

show abstract